Truncation at conserved terminal regions of BRCA1 protein is associated with highly proliferating hereditary breast cancers

Cancer Res. 1996 Jul 15;56(14):3216-9.

Abstract

The existence of two subgroups of BRCA1-associated breast cancer (BC) families has been recently posited: the first with highly proliferating tumors, and the second composed of cases with a low proliferation rate. Our aim was to test whether the proliferation rate of BRCA1-associated breast cancers was affected by the site of the germ line mutation in the BRCA1 gene. We analyzed the distribution of the mitotic index, a histoprognostic grade component shown to segregate in families, matching for germ line mutation location in a series of 28 breast cancers from 20 kindreds. We observed a prevalence of highly proliferating tumors when the mutation occurs in the two terminal conserved domains of the BRCA1 protein, ie., in the amino and carboxyl termini (P = 0.0024). Our data provide evidence for a genotype-phenotype correlation and along with their strong conservation during evolution argue for the importance of these two regions in the control of mammary cell growth.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Alleles
  • BRCA1 Protein
  • Breast Neoplasms / genetics*
  • Breast Neoplasms / pathology
  • Cell Cycle*
  • Female
  • Genes, Tumor Suppressor
  • Humans
  • Mutation
  • Neoplasm Proteins / physiology*
  • Ovarian Neoplasms / genetics
  • Sequence Deletion
  • Transcription Factors / physiology*

Substances

  • BRCA1 Protein
  • Neoplasm Proteins
  • Transcription Factors