Methylprednisolone prevents rejection of intrastriatal grafts of xenogeneic embryonic neural tissue in adult rats

Brain Res. 1996 Mar 18;712(2):199-212. doi: 10.1016/0006-8993(95)01409-8.

Abstract

We studied the effects of high-dose methylprednisolone on the survival of intrastriatal neural xenografts and the host responses against them. Dissociated mesencephalic tissue from inbred mouse (CBA-strain) embryos was transplanted to the intact striatum of adult Sprague-Dawley rats. The rats received either daily injections of methylprednisolone (30 mg/kg), or cyclosporin A (10 mg/kg), or no immunosuppressive treatment. Two or six weeks after transplantation, there was good survival of xenografts in both the methylprednisolone- and cyclosporin A-treated rats. In contrast, the xenografts in untreated control rats were all rejected by six weeks. There was no marked difference in the degree of expression of MHC class I and II antigens and the accumulation of activated astrocytes and microglial cells/macrophages between the three groups. However, both methylprednisolone and cyclosporin A reduced infiltration of T lymphocytes to the transplantation sites. The expression of pro-inflammatory cytokines (interferon-gamma, tumour necrosis factor-alpha, interleukin-6) in and around the grafts was lower in the methylprednisolone- and cyclosporin A-treated groups than in untreated control rats. Although high-dose methylprednisolone caused significant body weight loss, we conclude that this treatment can prevent rejection of intrastriatal grafts of xenogeneic embryonic neural tissue in the adult.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Brain Chemistry / drug effects
  • Brain Chemistry / physiology
  • Brain Tissue Transplantation / immunology*
  • CD4-CD8 Ratio / drug effects
  • Cytokines / biosynthesis
  • Female
  • Fetal Tissue Transplantation / immunology*
  • Glial Fibrillary Acidic Protein / metabolism
  • Graft Rejection / prevention & control*
  • Graft Survival / drug effects
  • Immunohistochemistry
  • Immunosuppressive Agents / therapeutic use*
  • Major Histocompatibility Complex / immunology
  • Methylprednisolone / therapeutic use*
  • Mice
  • Neostriatum / physiology*
  • Pregnancy
  • Rats
  • Rats, Sprague-Dawley
  • Receptors, Complement / immunology
  • Transplantation, Heterologous / immunology*
  • Tyrosine 3-Monooxygenase / metabolism

Substances

  • Cytokines
  • Glial Fibrillary Acidic Protein
  • Immunosuppressive Agents
  • Receptors, Complement
  • Tyrosine 3-Monooxygenase
  • Methylprednisolone