Central opiate modulation of peripheral IL-6 in rats

Neuroreport. 1996 Apr 26;7(6):1181-4. doi: 10.1097/00001756-199604260-00017.

Abstract

The effect of opiate agonists and antagonists on the induction of serum interleukin-6 (IL-6), an inflammatory cytokine that plays a major role in the acute phase response, was studied. Morphine (10 micrograms, i.c.v. or 15 mg kg-1, i.p.), beta-endorphin (0.5 or 5 micrograms, i.c.v.) and etorphin (10 mg kg-1, i.p.) induced IL-6. Moreover, morphine potentiated the IL-6 response induced by IL-1 beta (400 ng, i.p. or i.c.v.). When injected intraperitoneally, the opiate antagonist naloxone hydrochloride, antagonized the IL-6 response induced by either i.c.v. or i.p. IL-1 beta. This effect was not seen with naloxone methiodide, which does not cross the blood-brain barrier. The data show that central opiates are effective modulators of the inflammatory cytokine IL-6.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Etorphine / pharmacology
  • Injections, Intraperitoneal
  • Injections, Intraventricular
  • Interleukin-1 / pharmacology*
  • Interleukin-6 / biosynthesis*
  • Interleukin-6 / blood
  • Male
  • Morphine / pharmacology
  • Naloxone / pharmacology
  • Narcotics / pharmacology*
  • Rats
  • Rats, Sprague-Dawley
  • beta-Endorphin / pharmacology

Substances

  • Interleukin-1
  • Interleukin-6
  • Narcotics
  • Naloxone
  • Etorphine
  • beta-Endorphin
  • Morphine