Expression of the MAGE-1, -2, -3, -4, and -6 genes in non-squamous cell carcinoma lesions of the head and neck

Acta Otolaryngol. 1996 Jul;116(4):633-9. doi: 10.3109/00016489609137901.

Abstract

The messenger RNA level of several MAGE genes, some of which have been proven to encode tumor rejection antigens recognized by cytotoxic T lymphocytes, were examined in 41 benign and malignant lesions of the head and neck region. By a reverse transcription-polymerase chain reaction assay and Southern blot hybridization, MAGE-1, -2, -3, -4, and -6 genes were expressed in 25%, 41.7%, 33.3%, 8.3% and 33.3% of 12 non-squamous cell carcinomas, respectively. These tumors consisted of 6 papillary adenocarcinomas, 3 adenoid cystic carcinomas, 2 adenocarcinomas, and 1 mucoepidermoid tumor. Of 7 non-Hodgkin's lymphomas, one case from the oropharynx and 2 from the nasopharynx expressed for the MAGE-1 and MAGE-2 genes, respectively. In contrast, none of 12 benign tumors expressed any of these MAGE genes. Interestingly, of 10 other lesions including hyperplasia, keratosis, and ulcer, one histologically diagnosed as dysplasia expressed the MAGE-2, -3, -4, and -6 genes. These results suggest that the MAGE genes may be expressed in malignant tumors and precancerous lesions but not in benign tumors. In addition, non-squamous cell carcinomas may be suitable targets for specific immunotherapy against MAGE gene products.

MeSH terms

  • Adenocarcinoma, Papillary / genetics
  • Antigens, Neoplasm / genetics*
  • Blotting, Southern
  • Carcinoma / genetics*
  • Carcinoma / immunology
  • Carcinoma, Adenoid Cystic / genetics
  • DNA Primers
  • Gene Expression Regulation, Neoplastic
  • Head and Neck Neoplasms / genetics*
  • Head and Neck Neoplasms / immunology
  • Humans
  • Hyperplasia
  • Immunotherapy
  • In Situ Hybridization
  • Keratosis / genetics
  • Lymphoma, Non-Hodgkin / genetics
  • Melanoma-Specific Antigens
  • Neoplasm Proteins / genetics*
  • Oral Ulcer / genetics
  • Polymerase Chain Reaction
  • Precancerous Conditions / genetics*
  • Precancerous Conditions / immunology
  • RNA, Messenger / analysis
  • RNA, Messenger / genetics
  • T-Lymphocytes, Cytotoxic / immunology
  • Transcription, Genetic

Substances

  • Antigens, Neoplasm
  • DNA Primers
  • MAGEA1 protein, human
  • MAGEA3 protein, human
  • MAGEA4 protein, human
  • MAGEA6 protein, human
  • MAGEB2 protein, human
  • Melanoma-Specific Antigens
  • Neoplasm Proteins
  • RNA, Messenger