Regulation of NAD+ glycohydrolase activity by NAD(+)-dependent auto-ADP-ribosylation

Biochem J. 1996 Sep 15;318 ( Pt 3)(Pt 3):903-8. doi: 10.1042/bj3180903.

Abstract

NAD+ glycohydrolase (NADase; EC 3.2.2.5) is an enzyme that catalyses hydrolysis of NAD+ to produce ADP-ribose and nicotinamide. Its physiological role and the regulation of its enzymic activity have not been fully elucidated. In the present study, the mechanism of self-inactivation of NADase by its substrate, NAD+, was investigated by using intact rabbit erythrocytes and purified NADase. Our results suggest that inactivation of NADase was due an auto-ADP-ribosylation reaction. ADP-ribosylated NADase of rabbit erythrocytes was deADP-ribosylated when incubated without NAD+, and thus enzyme activity was simultaneously restored. These findings suggest that reversible auto-ADP-ribosylation of NADase might regulate the enzyme's activity in vivo.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenosine Diphosphate Ribose / metabolism*
  • Animals
  • Binding Sites
  • Cysteine / chemistry
  • Erythrocytes / enzymology
  • In Vitro Techniques
  • Kinetics
  • Molecular Weight
  • NAD / metabolism*
  • NAD+ Nucleosidase / antagonists & inhibitors
  • NAD+ Nucleosidase / chemistry
  • NAD+ Nucleosidase / metabolism*
  • Rabbits

Substances

  • NAD
  • Adenosine Diphosphate Ribose
  • NAD+ Nucleosidase
  • Cysteine