Nerve growth factor delays retinal degeneration in C3H mice

Graefes Arch Clin Exp Ophthalmol. 1996 Aug:234 Suppl 1:S96-100. doi: 10.1007/BF02343055.

Abstract

Background: The aim of the present study was to investigate the biological role of nerve growth factor (NGF) on retinal degeneration in the C3H mouse strain. This strain is characterized by a single gene mutation (rd) which leads to photoreceptor degeneration resembling human retinitis pigmentosa.

Methods: Neural retinas from 1- to 25 day-old C3H mice were dissected from outer ocular tissues, dissociated in cell suspension, stained with a vital dye and counted in a hemocytometer. For in vivo study, NGF was injected into the intraocular or retro-ocular area, and at the end of the treatment the mice were killed. The eyes were enucleated, fixed and cut by cryostat into 14-microns serial sections. The serial sections were stained with hematoxylin-eosin and the outer nuclear layer (ONL) was measured using a computerized image analysis system.

Results: An intraocular injection of NGF, or repeated retro-ocular injections, induced a significant increase in ONL thickness compared to controls.

Conclusion: Our data show that NGF inhibits retinal degeneration in C3H mice. The mechanism(s) underlying the protective action of NGF against retinal cell death remains to be established.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Division / drug effects
  • Cell Survival / drug effects
  • Cytochrome c Group / administration & dosage
  • Cytochrome c Group / pharmacology
  • Injections
  • Mice
  • Mice, Inbred C3H
  • Nerve Growth Factors / administration & dosage
  • Nerve Growth Factors / pharmacology*
  • Retina / drug effects*
  • Retina / pathology
  • Retinal Degeneration / pathology
  • Retinal Degeneration / prevention & control*
  • Vitreous Body

Substances

  • Cytochrome c Group
  • Nerve Growth Factors