Transforming growth factor-beta in the development of rat diabetic nephropathy. A 10-month study with insulin-treated rats

Nephron. 1996;74(1):189-96. doi: 10.1159/000189300.

Abstract

We investigated the intrarenal distribution of transforming growth factor-beta 1 (TGF-beta 1) protein and the TGF-beta 1 mRNA levels in the glomeruli and renal cortex of Wistar rats with streptozotocin-induced diabetes before and after the onset of diabetic nephropathy. Monthly urinary albumin excretion, glomerular filtration rate, glomerular volume, renal histology and immunohistochemical reaction for type-I collagen were also studied. The results showed progressively higher glomerular immunohistochemical TGF-beta 1 staining in rats with a diabetes duration of 24 and 40 weeks which was correlated with albuminuria (r = 0.905, p < 0.01) and was temporally associated with the appearance of glomerular deposition of total and type-I collagen. The glomerular content of TGF-beta 1 mRNA was higher in rats diabetic for 20 weeks while lower cortical RNA-TGF-beta 1 levels were found in rats with a diabetes duration of 1-40 weeks. These data suggest that this polypeptide may be an important mediator of diabetic glomerulosclerosis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Blotting, Northern
  • Diabetic Nephropathies / chemically induced
  • Diabetic Nephropathies / drug therapy
  • Diabetic Nephropathies / metabolism*
  • Female
  • Hyperglycemia / metabolism
  • Immunohistochemistry
  • Insulin / pharmacology*
  • Kidney Cortex / chemistry
  • Kidney Cortex / metabolism
  • Kidney Cortex / pathology
  • Kidney Function Tests
  • RNA, Messenger / analysis
  • Rats
  • Rats, Wistar
  • Sclerosis
  • Transforming Growth Factor beta / biosynthesis
  • Transforming Growth Factor beta / genetics
  • Transforming Growth Factor beta / metabolism*

Substances

  • Insulin
  • RNA, Messenger
  • Transforming Growth Factor beta