Selective loss of neurofilament proteins after exposure of differentiated human IMR-32 neuroblastoma cells to oxidative stress

Brain Res. 1996 Oct 28;738(1):162-6. doi: 10.1016/0006-8993(96)00992-4.

Abstract

Millimolar concentrations of ascorbate in the presence of iron can cause neuronal cell death. This study shows that the human neuronal cell line IMR-32 is sensitive to ascorbate and that cytotoxicity can be blocked by the antioxidant enzymes Cu/Zn-superoxide dismutase and catalase. There was a selective loss of neurofilament proteins after exposure to 5 or 10 mM ascorbate, as assessed by immunostaining and by Western blotting. Loss of actin or tubulin was not seen, suggesting that loss of neurofilaments is a sensitive and selective marker for free radical damage in these cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Ascorbic Acid / pharmacology
  • Blotting, Western
  • Catalase / pharmacology
  • Cell Death
  • Cell Differentiation
  • Dose-Response Relationship, Drug
  • Humans
  • Immunologic Techniques
  • Neurofilament Proteins / metabolism*
  • Oxidative Stress / physiology*
  • Staining and Labeling
  • Superoxide Dismutase / pharmacology
  • Time Factors
  • Tumor Cells, Cultured / drug effects
  • Tumor Cells, Cultured / pathology
  • Tumor Cells, Cultured / ultrastructure

Substances

  • Neurofilament Proteins
  • Catalase
  • Superoxide Dismutase
  • Ascorbic Acid