On the molecular mechanism of metmyoglobin-catalyzed reduction of hydrogen peroxide by ascorbate

Free Radic Biol Med. 1997;22(4):657-67. doi: 10.1016/s0891-5849(96)00382-6.

Abstract

Hydrogen peroxide induces rapid oxidation of metmyoglobin with an apparent second order rate constant, k1 = 3.4 x 10(4) M-1 min-1. The product of this interaction is ferrylmyoglobin with an unstable free radical on the globin moiety. This activated form of myoglobin is able: (a) to initiate the peroxidation of erythrocyte membranes and (b) to form intra- and intermolecular covalent crosslinkings. The presence of ascorbic acid in amounts stoichiometric to H2O2 efficiently prevents all the above processes. Moreover, in the presence of ascorbic acid a cyclic process is taking place leading to H2O2 reduction, ascorbic acid oxidation, and unmodified metmyoglobin formation (reaction 1).

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acetylation
  • Animals
  • Ascorbic Acid / pharmacology*
  • Free Radicals / metabolism
  • Horses
  • Hydrogen Peroxide / metabolism*
  • Hydrogen Peroxide / pharmacology
  • In Vitro Techniques
  • Kinetics
  • Metmyoglobin / drug effects
  • Metmyoglobin / metabolism*
  • Muscles / injuries
  • Muscles / metabolism
  • Oxidation-Reduction
  • Oxidative Stress
  • Reactive Oxygen Species / metabolism
  • Tyrosine / metabolism

Substances

  • Free Radicals
  • Reactive Oxygen Species
  • ferrylmyoglobin
  • Metmyoglobin
  • Tyrosine
  • Hydrogen Peroxide
  • Ascorbic Acid