[gamma-Glutamylcysteine synthetase and chemosensitivity]

Gan To Kagaku Ryoho. 1997 Feb;24(4):418-23.
[Article in Japanese]

Abstract

The gene transfectant with gamma-glutamylcysteine synthetase (gamma-GCS) gene, a rate limiting enzyme in GSH biosynthesis, exerted increased GSH content and ATP-dependent glutathione S-conjugate export pump (GS-X pump) decreased intracellular platinum and sensitivity against cisplatin. It is suggested that GS-X pump expression is related to cellular GSH metabolism and involved in cisplatin resistance. gamma-GCS transfectant also showed the resistance to MMC with the decreased DNA-MMC adduct. On the other hand, a MMC derivative, KW-2149, which is activated by thiol molecule, exhibited high cytotoxicity against the transfectant with an equal amount of DNA-MMC adduct compared with parental cells. It is suggested that KW-2149 could circumvent the GSH-mediated drug resistance.

Publication types

  • English Abstract
  • Review

MeSH terms

  • 3T3 Cells
  • Animals
  • Antineoplastic Agents / pharmacology*
  • Carcinoma, Small Cell / enzymology
  • Carcinoma, Small Cell / pathology*
  • Cisplatin / pharmacology*
  • Drug Resistance, Neoplasm
  • Drug Screening Assays, Antitumor
  • Glutamate-Cysteine Ligase / genetics*
  • Humans
  • Lung Neoplasms / enzymology
  • Lung Neoplasms / pathology*
  • Mice
  • Mitomycin / pharmacology*
  • Mitomycins
  • RNA, Messenger / metabolism*

Substances

  • Antineoplastic Agents
  • Mitomycins
  • RNA, Messenger
  • KW 2149
  • Mitomycin
  • Glutamate-Cysteine Ligase
  • Cisplatin