Selective induction of monocyte and not neutrophil-attracting chemokines after influenza A virus infection

J Exp Med. 1996 Sep 1;184(3):1191-6. doi: 10.1084/jem.184.3.1191.

Abstract

It is characteristic for virus infections that monocytes/macrophages and lymphocytes infiltrate infected tissue while neutrophils are absent. To understand the mechanisms selectively attracting mononuclear cells in viral diseases, we examined in an influenza A virus model the expression and regulation of chemokines as candidate molecules responsible for the immigration of leukocytes into inflamed tissue. After influenza A virus infection of human monocytes, a rapid expression of the mononuclear cell attracting CC-chemokine genes MIP-1, MCP-1, and RANTES occurred which was followed by the release of chemokine proteins. In striking contrast to CC-chemokines, the expression of the prototype neutrophil CXC-chemoattractants IL-8 and GRO-alpha was completely suppressed after influenza A infection. The release of other neutrophil chemotactic factors was excluded by microchemotaxis assays. These results suggest that the virus-specific induction of mononuclear cell-attracting chemokines accounts for the preferential influx of mononuclear leukocytes into virus-infected tissue.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Chemokine CCL2 / metabolism
  • Chemokine CCL4
  • Chemokine CCL5 / metabolism
  • Chemokines / blood*
  • Chemotaxis, Leukocyte
  • Humans
  • Influenza, Human / blood*
  • Lipopolysaccharides / pharmacology
  • Macrophage Inflammatory Proteins / metabolism
  • Monocytes / virology*
  • Neutrophils / virology*
  • Orthomyxoviridae

Substances

  • Chemokine CCL2
  • Chemokine CCL4
  • Chemokine CCL5
  • Chemokines
  • Lipopolysaccharides
  • Macrophage Inflammatory Proteins