Expression of serine/threonine kinase receptors in traumatic brain injury

Neuroreport. 1997 Jan 20;8(2):475-9. doi: 10.1097/00001756-199701200-00020.

Abstract

The aim of this study was to investigate the expression of serine/threonine kinase receptors in the brain following traumatic brain injury. We report here that, the recently cloned and characterized bone morphogenetic protein (BMP) receptor type II (BMPR-II) and the activin receptor type IA (ActR-1) Act: mRNAs were simultaneously up-regulated in neurones in the dentate gyrus 6 h after a mild cerebral contusion injury. This findings was specific for these receptors since other investigated genes (i.e. ActR-II, ActR-IB, trkB and c-fos) showed other temporal patterns. These data suggest that type I and type II receptors act together in signal transduction in vivo and that BMPs may be involved in neuronal plasticity after traumatic brain injury.

MeSH terms

  • Animals
  • Bone Morphogenetic Protein Receptors, Type II
  • Brain Injuries / metabolism*
  • In Situ Hybridization
  • Male
  • Protein Serine-Threonine Kinases / metabolism*
  • Rats
  • Rats, Sprague-Dawley
  • Receptors, Amino Acid / metabolism*
  • Uridine Monophosphate / analogs & derivatives*
  • Uridine Monophosphate / metabolism

Substances

  • Receptors, Amino Acid
  • 1-(5'-phospho-beta-D-ribofuranosyl)barbituric acid
  • Uridine Monophosphate
  • Protein Serine-Threonine Kinases
  • Bmpr2 protein, rat
  • Bone Morphogenetic Protein Receptors, Type II