omega-Conotoxin-sensitive Ca2+ voltage-gated channels modulate protein secretion in cultured rat Sertoli cells

Mol Cell Endocrinol. 1997 Feb 7;126(2):117-23. doi: 10.1016/s0303-7207(96)03973-1.

Abstract

Recent results have demonstrated that substantial calcium influx in rat Sertoli cells is mediated by cation channels of both L- and N-type. In this report we have investigated the possible role of such channels in the protein secretion of immature rat Sertoli cell monolayers. The blocking of N-type voltage-gated channels by omega-conotoxin (omega-CTX) GVIA results in a 50-60% inhibition of the protein secretion in the culture medium while total protein and RNA synthesis are not affected. The same extent of protein secretion inhibition is obtained in FSH-stimulated Sertoli cells. L-type voltage-gated channels apparently are not involved in such a modulation. These data, showing that a major fraction of secreted proteins from cultured rat Sertoli cells is Ca2+ dependent, represent the first evidence of a physiological role of voltage-operated Ca2+ channels in mammalian testis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Calcium Channels / metabolism*
  • Ion Channel Gating / drug effects
  • Male
  • Peptides / pharmacology
  • Proteins / metabolism*
  • Rats
  • Rats, Wistar
  • Sertoli Cells / metabolism*
  • omega-Conotoxin GVIA

Substances

  • Calcium Channels
  • Peptides
  • Proteins
  • omega-Conotoxin GVIA