The significance of polycystic-appearing ovaries versus normal-appearing ovaries in patients with polycystic ovary syndrome

Fertil Steril. 1997 Apr;67(4):631-5. doi: 10.1016/s0015-0282(97)81357-x.

Abstract

Objective: To evaluate what hormonal or biochemical parameters are most highly associated with the finding of polycystic-appearing ovaries as compared with normal-appearing ovaries in women with polycystic ovary syndrome (PCOS).

Design: Prospective randomized study.

Setting: Academic medical center.

Patients: Ten women with PCOS-five with normal-appearing ovaries and five with polycystic-appearing ovaries-were matched for age and body mass index. All had serum T levels between 80 and 150 ng/dL (conversion factor to SI unit, 3.467).

Intervention(s): Insulin infusion for the purpose of performing insulin tolerance testing to evaluate insulin resistance or sensitivity.

Main outcome measure(s): We measured serum T, DHEAS, androstenedione, sex-hormone binding globulin, 5 alpha-androstane-3 alpha-17 beta-androstenediol glucuronide, FSH, LH, insulin-like growth factor-I, insulin-like growth factor binding protein-1, and insulin-like growth factor binding protein-3. Insulin resistance, measured by insulin tolerance testing, also was done on the same day after the patient had fasted for at least 8 hours.

Result(s): Serum androgens and binding proteins were not significantly different in both groups. Insulin tolerance testing demonstrated a slower glucose disappearance in the polycystic appearing ovary group (Kitt glucose was 4.58% +/- 1.4%/min in the normal-appearing ovaries group versus 2.07% +/- 1.07%/min in the polycystic-appearing ovaries group).

Conclusion(s): Women with PCOS and polycystic-appearing ovaries do not demonstrate any definitive serum hormonal differences compared with women with PCOS and normal-appearing ovaries. The presence of polycystic-appearing ovaries correlates significantly with the presence of insulin resistance.

MeSH terms

  • Adult
  • Blood Glucose / metabolism
  • Carrier Proteins / blood
  • Cohort Studies
  • Female
  • Gonadal Steroid Hormones / blood
  • Gonadotropins, Pituitary / blood
  • Humans
  • Hypoglycemic Agents / administration & dosage
  • Hypoglycemic Agents / pharmacology
  • Insulin / administration & dosage
  • Insulin / pharmacology
  • Insulin Resistance / physiology*
  • Insulin-Like Growth Factor I / analysis
  • Ovary
  • Polycystic Ovary Syndrome / blood
  • Polycystic Ovary Syndrome / physiopathology*
  • Prospective Studies

Substances

  • Blood Glucose
  • Carrier Proteins
  • Gonadal Steroid Hormones
  • Gonadotropins, Pituitary
  • Hypoglycemic Agents
  • Insulin
  • Insulin-Like Growth Factor I