Role of endothelium-derived nitric oxide-endothelin balance in contrast medium-induced acute renal vasoconstriction in dogs

Acad Radiol. 1997 May;4(5):343-8. doi: 10.1016/s1076-6332(97)80115-8.

Abstract

Rationale and objectives: The authors evaluated the involvement of nitric oxide and endothelin in radiographic contrast medium-induced changes in renal hemodynamics.

Methods: Eleven anesthetized healthy dogs were each studied during three periods. Thirty minutes before the first, second, and third periods, the dogs received 1 mL per kilogram of body weight of isotonic saline, L-N-nitro-L-arginine-methyl-ester (L-Name, 10 mg/kg intravenously), and L-arginine (500 mg/ kg intravenously), respectively. Renal blood flow (RBF) and mean arterial blood pressure were continuously monitored. The glomerular filtration rate (GFR) was evaluated by means of polyfructosan clearance.

Results: Contrast medium induced a significant (P < .05) decrease in RBF and GFR and a significant (P < .05) increase in urinary endothelin excretion. L-Name enhanced the effect of contrast media on RBF and GFR. L-arginine attenuated the effect of L-Name on the contrast medium-induced reduction of GFR.

Conclusion: These findings support the hypothesis that acute contrast medium-induced intrarenal vasoconstriction may involve an imbalance of endothelial vasoactive agents, nitric oxide, and endothelin, and they confirm the involvement of hemodynamic changes in contrast medium-induced nephropathy.

MeSH terms

  • Animals
  • Arginine / pharmacology
  • Contrast Media / pharmacology*
  • Dogs
  • Endothelins / physiology*
  • Hemodynamics / drug effects
  • Kidney / drug effects
  • Kidney / physiology*
  • NG-Nitroarginine Methyl Ester / pharmacology
  • Nitric Oxide / physiology*
  • Vasoconstriction / drug effects
  • Vasoconstriction / physiology*

Substances

  • Contrast Media
  • Endothelins
  • Nitric Oxide
  • Arginine
  • NG-Nitroarginine Methyl Ester