The presence of genetic anticipation suggests that the molecular basis of familial primary pulmonary hypertension may be trinucleotide repeat expansion
Chest
.
1997 Jun;111(6 Suppl):82S-83S.
doi: 10.1378/chest.111.6_supplement.82s.
Authors
J E Loyd
1
,
B Slovis
,
J A Phillips 3rd
,
M G Butler
,
T M Foroud
,
P M Conneally
,
J H Newman
Affiliation
1
Division of Allergy, Pulmonary and Critical Care Medicine, Vanderbilt University Medical Center, Nashville, Tenn 37232-2650, USA.
PMID:
9184540
PMCID:
PMC6767907
DOI:
10.1378/chest.111.6_supplement.82s
No abstract available
Publication types
Research Support, U.S. Gov't, P.H.S.
MeSH terms
Adult
DNA / genetics
Female
Humans
Hypertension, Pulmonary / genetics*
Male
Trinucleotide Repeats / genetics*
Substances
DNA
Grants and funding
P01 HD030329/HD/NICHD NIH HHS/United States
T32 HL007123/HL/NHLBI NIH HHS/United States
HL 07123/HL/NHLBI NIH HHS/United States
HL 48164/HL/NHLBI NIH HHS/United States