Abstract
During humoral immune responses, B-lymphocyte activation is followed by differentiation along either the plasma cell pathway or the memory B-cell pathway. Recent studies suggest that CD40-CD40 ligand, OX-OX40 ligand, a group of cytokines and intracellular transcriptional factors may all contribute to B-lymphocyte differentiation control.
MeSH terms
-
Animals
-
B-Lymphocytes / cytology*
-
B-Lymphocytes / immunology*
-
B-Lymphocytes / metabolism
-
CD40 Antigens / metabolism
-
CD40 Ligand
-
Cell Differentiation
-
Humans
-
Immunologic Memory*
-
Interleukins / metabolism
-
Lymphocyte Activation
-
Membrane Glycoproteins / metabolism
-
OX40 Ligand
-
Plasma Cells / cytology*
-
Plasma Cells / immunology*
-
Plasma Cells / metabolism
-
Receptors, OX40
-
Receptors, Tumor Necrosis Factor / metabolism
-
Transcription Factors / metabolism
-
Tumor Necrosis Factor Receptor Superfamily, Member 7 / metabolism
Substances
-
CD40 Antigens
-
Interleukins
-
Membrane Glycoproteins
-
OX40 Ligand
-
Receptors, OX40
-
Receptors, Tumor Necrosis Factor
-
TNFRSF4 protein, human
-
TNFSF4 protein, human
-
Transcription Factors
-
Tumor Necrosis Factor Receptor Superfamily, Member 7
-
CD40 Ligand