The activation-dependent induction of APN-(CD13) in T-cells is controlled at different levels of gene expression

FEBS Lett. 1997 Jul 21;412(1):53-6. doi: 10.1016/s0014-5793(97)00738-2.

Abstract

Recently, it was shown that aminopeptidase N (E.C. 3.4.11.2, CD13) is up-regulated during mitogenic stimulation of peripheral T-cells. In this study, we demonstrate that the half-life of APN mRNA was considerably prolonged in these cells leading to a 2.7-fold increase of APN transcript level. The apparent half-life time of the APN transcript was investigated by the RNA synthesis inhibitor-chase method using actinomycin D. The steady-state APN mRNA levels was determined by a competitive RT-PCR. The half-lives estimated in resting T-cells, natural killer cells and permanently growing tumour cells varied between 3.5 and 6 h. Finally, nuclear run-on assays revealed that the APN gene expression of stimulated T-cells is controlled by increased promoter activity as well. These studies suggest a control of APN gene expression at the post-transcriptional level in addition to promoter-mediated regulation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Binding, Competitive
  • CD13 Antigens / biosynthesis*
  • CD13 Antigens / genetics
  • Cell Line
  • Dactinomycin / pharmacology
  • Enzyme Activation
  • Enzyme Induction
  • Enzyme Stability
  • Half-Life
  • Kinetics
  • Polymerase Chain Reaction
  • Promoter Regions, Genetic
  • RNA, Messenger / metabolism
  • T-Lymphocytes / enzymology*

Substances

  • RNA, Messenger
  • Dactinomycin
  • CD13 Antigens