Resistance to apoptosis induced by serum depletion and all-trans retinoic acid in drug-resistant leukemic cell lines

Leuk Lymphoma. 1997 Jul;26(3-4):369-76. doi: 10.3109/10428199709051787.

Abstract

The relation between resistance to anticancer drugs and resistance to apoptosis has been investigated in the human leukemic cell line(KY-821) and its drug-resistant sublines. Under serum depletion conditions, drug-resistant cell lines showed apoptotic resistance when compared with the parental cell line. Drug resistant cell lines also showed resistance to apoptosis when treated with all-trans retinoic acid. DNA fragmentation was low in drug resistant cell lines under both stimulations. Flowcytometry analysis did not show any alterations of the Fas antigen, p53, bcl-2 and c-myc protein expression toward inhibition of apoptotic response in drug-resistant sublines. These results indicate that drug-resistant leukemic cells still show resistance to apoptosis-inducing stimulation such as poor nutrition and differentiation-inducing agents.

MeSH terms

  • Antineoplastic Agents / pharmacology*
  • Apoptosis / drug effects*
  • Blotting, Western
  • Culture Media, Serum-Free
  • DNA, Neoplasm / metabolism
  • Drug Resistance, Neoplasm
  • Electrophoresis
  • Humans
  • Leukemia / drug therapy*
  • Leukemia / metabolism*
  • Leukemia / pathology
  • Proto-Oncogene Proteins c-bcl-2 / biosynthesis
  • Proto-Oncogene Proteins c-myc / biosynthesis
  • Tretinoin / pharmacology*
  • Tumor Cells, Cultured
  • Tumor Suppressor Protein p53 / biosynthesis
  • fas Receptor / biosynthesis

Substances

  • Antineoplastic Agents
  • Culture Media, Serum-Free
  • DNA, Neoplasm
  • Proto-Oncogene Proteins c-bcl-2
  • Proto-Oncogene Proteins c-myc
  • Tumor Suppressor Protein p53
  • fas Receptor
  • Tretinoin