Anti-sense oligonucleotides directed against EGF-related growth factors enhance anti-proliferative effect of conventional anti-tumor drugs in human colon-cancer cells

Int J Cancer. 1997 Oct 9;73(2):277-82. doi: 10.1002/(sici)1097-0215(19971009)73:2<277::aid-ijc19>3.0.co;2-c.

Abstract

We have demonstrated that anti-sense phosphorothioate oligodeoxynucleotides (AS S-oligos) directed against the EGF-like growth factors CRIPTO (CR), amphiregulin (AR) or transforming-growth-factor-alpha(TGFalpha) mRNA, are equipotent in their ability to inhibit the growth of human colon-carcinoma GEO cells. In this study, we evaluated the effect of combinations of these AS S-oligos and conventional anti tumor drugs, such as 5-fluorouracil (5-FU), adriamycin (ADR), mitomycin C (MIT) and cis-platinum (CDDP), on GEO cell growth. Dose-dependent growth inhibition was observed by treatment either with AS S-oligos or with anti-tumor drugs, using a clonogenic assay. Furthermore, an additive growth inhibitory effect occurred when GEO cells were exposed to the AS S-oligos after treatment with different concentrations of either 5-FU, MIT, ADR or CDDP. For example, treatment of GEO cells with a combination of low concentrations of 5-FU and any of the 3 AS S-oligos resulted in up to 70% growth inhibition. However, treatment of GEO cells with AS S-oligos before exposure to 5-FU or CDDP resulted in reduced efficacy of both drugs. Flow-cytometric analysis of DNA content demonstrated that treatment with the AS S-oligos caused a slight reduction of the percentage of cells in the S-phase of the cell cycle. These data suggest that combinations of AS S-oligos directed against EGF-related growth factors and of conventional anti-tumor drugs may result in efficient inhibition of colon-carcinoma cell growth.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amphiregulin
  • Antineoplastic Agents / pharmacology*
  • Cell Division / drug effects
  • Cisplatin / pharmacology
  • Colonic Neoplasms / drug therapy*
  • Colonic Neoplasms / pathology
  • DNA, Neoplasm / drug effects
  • Doxorubicin / pharmacology
  • Drug Combinations
  • Drug Synergism
  • EGF Family of Proteins
  • Epidermal Growth Factor*
  • Flow Cytometry
  • Fluorouracil / pharmacology
  • GPI-Linked Proteins
  • Glycoproteins / genetics*
  • Growth Substances / genetics*
  • Humans
  • Intercellular Signaling Peptides and Proteins*
  • Membrane Glycoproteins*
  • Mitomycin / pharmacology
  • Neoplasm Proteins / genetics*
  • Oligonucleotides, Antisense / pharmacology*
  • RNA, Messenger / genetics
  • Thionucleotides / pharmacology
  • Transforming Growth Factor alpha / genetics*
  • Tumor Cells, Cultured

Substances

  • AREG protein, human
  • Amphiregulin
  • Antineoplastic Agents
  • DNA, Neoplasm
  • Drug Combinations
  • EGF Family of Proteins
  • GPI-Linked Proteins
  • Glycoproteins
  • Growth Substances
  • Intercellular Signaling Peptides and Proteins
  • Membrane Glycoproteins
  • Neoplasm Proteins
  • Oligonucleotides, Antisense
  • RNA, Messenger
  • TDGF1 protein, human
  • Thionucleotides
  • Transforming Growth Factor alpha
  • Mitomycin
  • Epidermal Growth Factor
  • Doxorubicin
  • Cisplatin
  • Fluorouracil