NF-M trans-activates the human DNA topoisomerase II alpha promoter independently of c-Myb in HL-60 cells

Leuk Res. 1997 Aug;21(8):711-20. doi: 10.1016/s0145-2126(97)00040-4.

Abstract

Identifying transcriptional regulators of DNA topoisomerase II alpha (topo II alpha) is essential to decipher the mechanisms underlying leukemia cell resistance to topo II-directed antitumor drugs. We have previously reported that the proto-oncogene transcription factor c-Myb transactivates the topo II alpha promoter in several hematopoietic cell lines. Currently, we investigate whether NF-M, a C/EBP beta family member, cooperates with c-Myb in activating topo II alpha transcription. Although NF-M is the most efficacious trans-activator of topo II alpha that we have examined (approximately 38-fold over basal), NF-M does not appear to be involved in the endogenous transcriptional regulation of topo II alpha. Interestingly, we report that the sodium butyrate-dependent induction of the topo II alpha promoter observed previously appears to be mediated by c-Myb, independent of NF-M.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Antigens, Neoplasm
  • CCAAT-Enhancer-Binding Proteins
  • COS Cells
  • DNA Topoisomerases, Type II* / genetics*
  • DNA-Binding Proteins / pharmacology*
  • Dose-Response Relationship, Drug
  • HL-60 Cells
  • HeLa Cells
  • Humans
  • Isoenzymes / genetics*
  • Nuclear Proteins / pharmacology*
  • Promoter Regions, Genetic
  • Proto-Oncogene Mas
  • Transcription Factors
  • Transcriptional Activation*

Substances

  • Antigens, Neoplasm
  • CCAAT-Enhancer-Binding Proteins
  • DNA-Binding Proteins
  • Isoenzymes
  • MAS1 protein, human
  • Nuclear Proteins
  • Proto-Oncogene Mas
  • Transcription Factors
  • DNA Topoisomerases, Type II