Deficient cell cycle control in myeloid cells of patients with newly diagnosed chronic myeloid leukemia

Cytokines Mol Ther. 1995 Dec;1(4):281-8.

Abstract

Cell cycle control subsequent to gamma irradiation or growth factor starvation has been studied in immative hematopoietic cells of 19 previously untreated chronic myeloid leukemia (CML) patients in chronic phase compared with 18 normal controls. CD34-positive cells were cultured for seven days in the presence of optimal concentrations of appropriate growth factors. At day 7 of culture both S-phase fraction and differentiation were identical in normal and leukemic cells. In normal cells the proportion of S-phase cells was reduced by irradiation with 500 rad from 40 +/- 3% to 16 +/- 2%. In contrast, in CML cells a reduction of S-phase cells from 35 +/- 2% to 25 +/- 3% was observed. Moreover, irradiated CML cells arrested at a smaller number of cells in G2. Similarly, a significantly higher proportion of CML cells remained in S phase after withdrawal of growth factors. Semiquantitative PCR of p21 (waf1/cip1) induction by gamma irradiation provided no evidence for a major functional deficiency of p53 response to irradiation in these cells. Our results demonstrate an abnormal cell cycle arrest in chronic-phase CML cells both after gamma irradiation and after growth factor removal. This observation might have important implications for understanding the pathogenesis of both hyperplasia of chronic phase and the development of blast crisis in CML. The molecular mechanisms underlying these abnormalities in bcr-abl-positive cells remain to be clarified.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Actins / biosynthesis
  • Antigens, CD / analysis
  • Antigens, CD34 / analysis
  • Bone Marrow / pathology
  • Cell Cycle / physiology*
  • Cell Cycle / radiation effects
  • Cells, Cultured
  • Cyclin-Dependent Kinase Inhibitor p21
  • Cyclins / biosynthesis
  • DNA Damage
  • DNA, Neoplasm / biosynthesis
  • GADD45 Proteins
  • Gamma Rays
  • Hematopoietic Stem Cells / pathology*
  • Hematopoietic Stem Cells / radiation effects
  • Humans
  • Intracellular Signaling Peptides and Proteins
  • Leukemia, Myelogenous, Chronic, BCR-ABL Positive / blood
  • Leukemia, Myelogenous, Chronic, BCR-ABL Positive / pathology*
  • Polymerase Chain Reaction
  • Protein Biosynthesis
  • Proteins*
  • Time Factors
  • Tumor Suppressor Protein p53 / biosynthesis

Substances

  • Actins
  • Antigens, CD
  • Antigens, CD34
  • CDKN1A protein, human
  • Cyclin-Dependent Kinase Inhibitor p21
  • Cyclins
  • DNA, Neoplasm
  • Intracellular Signaling Peptides and Proteins
  • Proteins
  • Tumor Suppressor Protein p53