P53 gene product and EGF-receptor are highly expressed in placental site trophoblastic tumor

Hum Pathol. 1997 Nov;28(11):1302-6. doi: 10.1016/s0046-8177(97)90206-9.

Abstract

Immunohistochemical analysis of curettage material from a placental site trophoblastic tumor (PSTT) revealed a high expression of p53 gene products, of epidermal growth factor receptor (EGF-R) and of Ki-67 (MIB-1) proliferation associated antigen. bcl-2 was not expressed. These results show that in PSTT inactivation/dysregulation of p53 and upregulation of EGF-R and MiB-1 occurs, indicating that these factors are probably involved in tumor genesis and propagation of PSTT. The prognostic significance of the molecular genetic data, however, remains to be established.

Publication types

  • Case Reports

MeSH terms

  • Adult
  • Biomarkers / analysis
  • Chorionic Gonadotropin / analysis
  • ErbB Receptors / analysis*
  • Female
  • Humans
  • Immunohistochemistry
  • Ki-67 Antigen / analysis
  • Placental Lactogen / analysis
  • Pregnancy
  • Trophoblastic Tumor, Placental Site / chemistry*
  • Tumor Suppressor Protein p53 / analysis*
  • Uterine Neoplasms / chemistry*

Substances

  • Biomarkers
  • Chorionic Gonadotropin
  • Ki-67 Antigen
  • Tumor Suppressor Protein p53
  • Placental Lactogen
  • ErbB Receptors