Tumour-derived, endocrine, exogenous and therapeutic factors differentially modulate cytokine secretion in whole blood cell culture

Eur J Cancer. 1997 Sep;33(10):1661-7. doi: 10.1016/s0959-8049(97)00159-7.

Abstract

Following our previous results which showed that TGF-beta 1 suppressed the secretion of certain cytokines, we investigated the effects of different endogenous and exogenous factors on cytokine secretion in whole blood cell culture by using an enzyme-linked immunosorbent assay (ELISA) for measurement of cytokine concentrations. Several molecules including dexamethasone, noradrenaline (NA) and ethanol differentially inhibited mitogen-induced cytokine secretion. Dexamethasone and noradrenaline suppressed secretion of IL-2, IFN alpha, IFN gamma, TNF alpha, IL-1 alpha and IL-1 beta. beta-Endorphin and Leu-Enkephalin had no significant influence on cytokine secretion. Suppression of cytokine secretion by TGF-beta 1 was further intensified significantly and dose dependently by addition of noradrenaline. GM-CSF stimulated the secretion of IL-1 alpha, IL-1 beta and TNF gamma, but had no influence on the secretion of IL-2, IFN alpha and IFN gamma. G-CSF, IL-3 and SCF did not significantly influence secretion of all cytokines tested. Thus, endogenous and exogenous factors differentially influence cytokine secretion by immunocompetent cells.

MeSH terms

  • Adult
  • Cell Culture Techniques
  • Colony-Stimulating Factors / pharmacology
  • Cytokines / blood*
  • Dexamethasone / pharmacology
  • Ethanol / pharmacology
  • Humans
  • Immune Tolerance / drug effects*
  • Immune Tolerance / immunology
  • Immunosuppressive Agents / pharmacology*
  • Neoplasms / immunology*
  • Norepinephrine / pharmacology
  • Transforming Growth Factor beta / pharmacology

Substances

  • Colony-Stimulating Factors
  • Cytokines
  • Immunosuppressive Agents
  • Transforming Growth Factor beta
  • Ethanol
  • Dexamethasone
  • Norepinephrine