Cisplatin-stimulated murine bone marrow-derived macrophages secrete oncostatin M

Immunol Cell Biol. 1997 Oct;75(5):492-6. doi: 10.1038/icb.1997.76.

Abstract

Cisplatin (CP), a widely used anticancer drug activates cells of the immune system to a tumoricidal state, and thus functions as a potent biological response modifier. Expression of oncostatin M (OSM), a novel cytokine having a growth regulatory effect, was studied in bone marrow-derived macrophages treated with cisplatin. Supernatants from CP-stimulated macrophages were found to be cytostatic for OSM-sensitive A375 melanoma cells. Immunoblot analysis with anti-OSM antibody revealed that expression of OSM in macrophages upon CP stimulation is a rapid process and within 30 min of CP treatment, a significant amount of OSM is secreted into the culture supernatant. These results therefore indicate that CP can stimulate murine bone marrow-derived macrophages to produce OSM which can be implicated as one of the cytostatic/ cytocidal factors in the antitumour action of cisplatin-stimulated macrophages.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antineoplastic Agents / pharmacology*
  • Bone Marrow Cells / cytology*
  • Bone Marrow Cells / drug effects
  • Cell Survival
  • Cells, Cultured
  • Cisplatin / pharmacology*
  • Growth Inhibitors / analysis
  • Growth Inhibitors / metabolism*
  • Luminescent Measurements
  • Macrophages / drug effects*
  • Macrophages / metabolism*
  • Mice
  • Mice, Inbred BALB C
  • Oncostatin M
  • Peptides / analysis
  • Peptides / metabolism*
  • Tumor Cells, Cultured

Substances

  • Antineoplastic Agents
  • Growth Inhibitors
  • Osm protein, mouse
  • Peptides
  • Oncostatin M
  • Cisplatin