Central nervous system TNFalpha-mRNA expression during rabbit experimental pneumococcal meningitis

Neurosci Lett. 1997 Nov 21;237(2-3):105-8. doi: 10.1016/s0304-3940(97)00830-6.

Abstract

In pneumococcal meningitis inflammatory mediators such as tumor necrosis factor alpha (TNFalpha) are produced in large quantities and play a major role in pathogenesis. It is not known exactly which cells produce these mediators during infection. We investigated the localisation of TNFalpha-mRNA in the central nervous system (CNS) by in situ hybridisation during experimental Streptococcus pneumoniae meningitis. TNF-positive cells were detected only in inflammatory infiltrates within the meninges. Cells within the brain parenchyma and the choroid plexus were completely negative. After monocyte depletion, no TNFalpha-mRNA positive cells were detected in the CNS. These findings suggest that TNFalpha in pneumococcal meningitis is produced in the CNS mainly by blood-derived, infiltrating monocytes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Central Nervous System / metabolism*
  • Central Nervous System / pathology
  • In Situ Hybridization
  • Meningitis, Pneumococcal / cerebrospinal fluid
  • Meningitis, Pneumococcal / metabolism*
  • Meningitis, Pneumococcal / pathology
  • Monocytes / metabolism
  • RNA, Messenger / biosynthesis*
  • RNA, Messenger / cerebrospinal fluid
  • Rabbits
  • Tumor Necrosis Factor-alpha / biosynthesis*
  • Tumor Necrosis Factor-alpha / cerebrospinal fluid

Substances

  • RNA, Messenger
  • Tumor Necrosis Factor-alpha