Acute oxygen supplementation does not relieve the impairment of respiratory muscle strength in hypoxemic COPD

Chest. 1998 Feb;113(2):334-9. doi: 10.1378/chest.113.2.334.

Abstract

Study objectives: To verify whether hypoxemia affects respiratory muscle strength in the absence of malnutrition and whether such effect, if present, is reversible with an acute oxygen supplementation.

Design: Case series analysis, before-after trial.

Setting: Outpatient pneumology departments of two university hospitals.

Patients: One hundred twenty patients affected by COPD in stable conditions having actual to ideal body weight ratio of > or =90%.

Measurements and results: Maximal inspiratory pressure (MIP) was measured at functional residual capacity level in the whole sample of subjects and during oxygen supplementation in 58 patients having a PaO2< or =60 mm Hg when breathing in room air. Predictors of MIP were assessed by a multivariate analysis. MIP values before and after oxygen supplementation were compared by a paired t test. MIP was independently correlated with FVC (p<0.001), PaO2 (p<0.01), and age (p<0.01). In the subgroup of hypoxemic patients, MIP values did not change significantly after oxygen supplementation (3.08+/-1.74 vs 3.03+/-1.91 kPa, t=0.43, not significant).

Conclusions: Hypoxemia is an important negative correlate of MIP even in well-nourished COPD patients. Its effect is not reversible with an acute oxygen supplementation.

Publication types

  • Comparative Study

MeSH terms

  • Age Factors
  • Aged
  • Body Weight
  • Female
  • Follow-Up Studies
  • Forecasting
  • Functional Residual Capacity / physiology
  • Humans
  • Hypoxia / physiopathology*
  • Hypoxia / therapy
  • Inhalation / physiology
  • Lung Diseases, Obstructive / physiopathology*
  • Lung Diseases, Obstructive / therapy
  • Male
  • Multivariate Analysis
  • Muscle Contraction / physiology*
  • Nutrition Disorders
  • Nutritional Status
  • Oxygen / blood
  • Oxygen Inhalation Therapy*
  • Pressure
  • Regression Analysis
  • Residual Volume / physiology
  • Respiratory Muscles / physiopathology*
  • Total Lung Capacity / physiology
  • Vital Capacity / physiology

Substances

  • Oxygen