The glycoprotein D (US6) homolog is not essential for oncogenicity or horizontal transmission of Marek's disease virus

J Virol. 1998 Mar;72(3):2548-53. doi: 10.1128/JVI.72.3.2548-2553.1998.

Abstract

RB1BUS6lacgpt, a Marek's disease virus (MDV) mutant having a disrupted glycoprotein D (gD) homolog gene, established infection and induced tumors in chickens exposed to it by inoculation or by contact. Lymphoblastoid cell lines derived from RB1BUS6lacgpt-induced tumors harbored only the mutant virus. These results provide strong evidence that an intact gD homolog gene is not essential for oncogenicity or horizontal transmission of MDV.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Animals
  • Bacterial Proteins / genetics
  • Cell Transformation, Viral*
  • Chickens
  • Cloning, Molecular
  • Disease Transmission, Infectious*
  • Escherichia coli Proteins
  • Gene Expression
  • Herpesvirus 2, Gallid / genetics
  • Herpesvirus 2, Gallid / isolation & purification
  • Herpesvirus 2, Gallid / metabolism
  • Herpesvirus 2, Gallid / pathogenicity*
  • Lac Operon
  • Lymphoma
  • Marek Disease / transmission*
  • Marek Disease / virology
  • Mutagenesis, Insertional
  • Pentosyltransferases
  • Proteins*
  • RNA, Viral
  • Tumor Cells, Cultured
  • Viral Envelope Proteins / genetics
  • Viral Envelope Proteins / physiology*

Substances

  • Bacterial Proteins
  • Escherichia coli Proteins
  • Proteins
  • RNA, Viral
  • Viral Envelope Proteins
  • glycoprotein D, Marek's disease virus
  • Pentosyltransferases
  • Gpt protein, E coli