Hypoxia-induced free iron release in the red cells of newborn infants

Acta Paediatr. 1998 Jan;87(1):77-81. doi: 10.1080/08035259850157912.

Abstract

Heparinized blood samples were obtained at birth from 164 newborn infants (101 full term; 63 preterm). Intra-erythrocyte free iron concentration and hypoxanthine plasma levels were determined by high-pressure liquid chromatography. Intra-erythrocyte free iron concentration was higher in preterm than in full term babies (p < 0.0001) and adults (p < 0.0001). Statistically significant correlations were observed between intra-erythrocyte free iron concentration and hypoxanthine levels (r = 0.66; p = 0.0001), pH (r = -0.76; p = 0.0001), base excess (r = -0.79; p = 0.0001), and gestational age (r = -0.44; p = 0.0001) in both infant populations. Multiple regression analysis between intra-erythrocyte free iron concentration in cord blood, as an independent variable, and Apgar score at 1 min, pH, base excess, hypoxanthine values, FiO2 needed for resuscitation immediately after delivery, and gestational age, as dependent variables, identified hypoxanthine levels (p = 0.0003; partial F-test = 15.4) as the best single predictor of intra-erythrocyte free iron concentration. In conclusion, hypoxia induces intra-erythrocyte free iron release, and therefore enhances the risk of oxidative injury due to hydroxyl radical generation.

Publication types

  • Clinical Trial
  • Controlled Clinical Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Erythrocytes / metabolism*
  • Female
  • Ferritins / blood*
  • Fetal Blood / metabolism*
  • Humans
  • Hypoxia / blood
  • Hypoxia / complications*
  • Infant, Newborn
  • Infant, Premature / blood*
  • Linear Models
  • Male
  • Oxidative Stress
  • Reactive Oxygen Species / metabolism*
  • Reference Values
  • Regression Analysis

Substances

  • Reactive Oxygen Species
  • Ferritins