Low plasma ascorbic acid independently predicts the presence of an unstable coronary syndrome

J Am Coll Cardiol. 1998 Apr;31(5):980-6. doi: 10.1016/s0735-1097(98)00059-x.

Abstract

Objectives: This study sought to investigate the relations between plasma antioxidant status, extent of atherosclerosis and activity of coronary artery disease.

Background: Previous studies indicate that increased antioxidant intake is associated with decreased coronary disease risk, but the underlying mechanisms remain controversial.

Methods: Plasma samples were obtained from 149 patients undergoing cardiac catheterization (65 with stable angina, 84 with unstable angina or a myocardial infarction within 2 weeks). Twelve plasma antioxidant/oxidant markers were measured and correlated with the extent of atherosclerosis and the presence of an unstable coronary syndrome.

Results: By multiple linear regression analysis, age (p < 0.001), diabetes mellitus (p < 0.001), male gender (p < 0.001) and hypercholesterolemia (p = 0.02) were independent predictors of the extent of atherosclerosis. No antioxidant/oxidant marker correlated with the extent of atherosclerosis. However, lower plasma ascorbic acid concentration predicted the presence of an unstable coronary syndrome by multiple logistic regression (odds ratio [OR] 0.59, 95% confidence interval [CI] 0.40 to 0.89, p = 0.01). The severity of atherosclerosis also predicted the presence of an unstable coronary syndrome (OR 1.7, 95% CI 1.14 to 2.47, p = 0.008) when all patients were considered. When only patients with significant coronary disease were considered (at least one stenosis >50%), ascorbic acid concentration (OR 0.56, 95% CI 0.37 to 0.85, p = 0.008) and total plasma thiols (OR 0.52, 95% CI 0.34 to 0.80, p = 0.004) predicted the presence of an unstable coronary syndrome, whereas the extent of atherosclerosis did not.

Conclusions: These data are consistent with the hypothesis that the beneficial effects of antioxidants in coronary artery disease may result, in part, by an influence on lesion activity rather than a reduction in the overall extent of fixed disease.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Aged
  • Angina Pectoris / blood
  • Angina, Unstable / blood
  • Antioxidants
  • Arteriosclerosis / blood
  • Ascorbic Acid / blood*
  • Biomarkers / blood
  • Coronary Disease / blood*
  • Female
  • Humans
  • Linear Models
  • Lipid Peroxidation*
  • Logistic Models
  • Male
  • Middle Aged
  • Myocardial Infarction / blood

Substances

  • Antioxidants
  • Biomarkers
  • Ascorbic Acid