A signaling complex of Ca2+-calmodulin-dependent protein kinase IV and protein phosphatase 2A

Science. 1998 May 22;280(5367):1258-61. doi: 10.1126/science.280.5367.1258.

Abstract

Stimulation of T lymphocytes results in a rapid increase in intracellular calcium concentration ([Ca2+]i) that parallels the activation of Ca2+-calmodulin-dependent protein kinase IV (CaMKIV), a nuclear enzyme that can phosphorylate and activate the cyclic adenosine monophosphate (cAMP) response element-binding protein (CREB). However, inactivation of CaMKIV occurs despite the sustained increase in [Ca2+]i that is required for T cell activation. A stable and stoichiometric complex of CaMKIV with protein serine-threonine phosphatase 2A (PP2A) was identified in which PP2A dephosphorylates CaMKIV and functions as a negative regulator of CaMKIV signaling. In Jurkat T cells, inhibition of PP2A activity by small t antigen enhanced activation of CREB-mediated transcription by CaMKIV. These findings reveal an intracellular signaling mechanism whereby a protein serine-threonine kinase (CaMKIV) is regulated by a tightly associated protein serine-threonine phosphatase (PP2A).

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Antigens, Polyomavirus Transforming / metabolism
  • Brain / enzymology
  • Calcium / metabolism
  • Calcium-Calmodulin-Dependent Protein Kinase Type 4
  • Calcium-Calmodulin-Dependent Protein Kinases / genetics
  • Calcium-Calmodulin-Dependent Protein Kinases / isolation & purification
  • Calcium-Calmodulin-Dependent Protein Kinases / metabolism*
  • Calmodulin / metabolism
  • Coenzymes / metabolism
  • Cyclic AMP Response Element-Binding Protein / metabolism
  • Enzyme Activation
  • Humans
  • Jurkat Cells
  • Lymphocyte Activation
  • Mutation
  • Phosphoprotein Phosphatases / isolation & purification
  • Phosphoprotein Phosphatases / metabolism*
  • Phosphorylation
  • Protein Phosphatase 2
  • Rats
  • Recombinant Fusion Proteins / metabolism
  • Signal Transduction*
  • T-Lymphocytes / enzymology*
  • Transcription, Genetic

Substances

  • Antigens, Polyomavirus Transforming
  • Calmodulin
  • Coenzymes
  • Cyclic AMP Response Element-Binding Protein
  • Recombinant Fusion Proteins
  • CAMK4 protein, human
  • Calcium-Calmodulin-Dependent Protein Kinase Type 4
  • Calcium-Calmodulin-Dependent Protein Kinases
  • Camk4 protein, rat
  • Phosphoprotein Phosphatases
  • Protein Phosphatase 2
  • Calcium