Gene-targeted mice reveal importance of L-selectin-dependent rolling for neutrophil adhesion

Am J Physiol. 1998 May;274(5):H1785-91. doi: 10.1152/ajpheart.1998.274.5.H1785.

Abstract

It has not been determined whether L-selectin-mediated rolling can promote leukocyte adhesion in vivo independent of P- and E-selectin. We used intravital microscopy of E- and P-selectin double-mutant mice (E-/P-) stimulated with tumor necrosis factor-alpha for 6-8 h to investigate the importance of L-selectin-dependent rolling in cremaster muscle venules. Rolling leukocyte flux in E-/P- mice was 9 +/- 2 cells/min compared with 77 +/- 17 cells/min in wild-type (WT) mice. Pretreatment with the L-selectin monoclonal antibody MEL-14 significantly reduced rolling in both E-/P- (by 89%) and WT mice (by 79%). L-selectin-dependent rolling in E-/P- mice resulted in leukocyte adhesion comparable to that seen in WT mice. MEL-14 pretreatment of E-/P- mice reduced leukocyte adhesion by 50%. The majority (approximately 80%) of intravascular leukocytes in both WT and E-/P- mice were neutrophils. We conclude that L-selectin can mediate rolling that results in sufficient leukocyte recruitment to account for the robust inflammatory response seen in E-/P- mice at later times.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Cell Adhesion / physiology
  • E-Selectin / physiology*
  • Gene Targeting
  • Inflammation / pathology
  • Inflammation / physiopathology
  • L-Selectin / physiology*
  • Leukocytes / pathology*
  • Leukocytes / physiology*
  • Mice
  • Mice, Knockout
  • Neutrophil Activation / physiology
  • Neutrophils / pathology*
  • Neutrophils / physiology*
  • P-Selectin / physiology*

Substances

  • E-Selectin
  • P-Selectin
  • L-Selectin