Circulating endotoxin during initial antibiotic treatment of severe gram-negative bacteremic infections

J Infect Dis. 1998 Jul;178(1):270-3. doi: 10.1086/517451.

Abstract

The impact of antibiotics on total endotoxemia and circulating tumor necrosis factor (TNF)-alpha, interleukin (IL)-6, and IL-8 in 18 patients with severe bacteremic sepsis or septic shock due to gram-negative species was investigated. Endotoxemia, TNF-alpha, IL-6, and IL-8 were assayed before (H0) and 1 h (H1) and 4 h (H4) after the first antibiotic infusion. Endotoxemia decreased from H0 (median, 0.4 EU/mL; interquartile interval, 0.09-1.23) to H1 (median, 0.19 EU/mL; interquartile interval, 0.07-0.75; P = .03) and remained stable between H1 and H4 (median, 0.12 EU/mL; interquartile interval, 0.09-0.30; P = .4). IL-6 levels fell between H0 and H4 (P = .01) and between H1 and H4 (P = .03). IL-8 was higher at H0 than at H1 (P = .04) and at H4 (P = .01). These results suggest that endotoxemia is not increased by antibiotherapy of severe gram-negative bacteremia.

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • Anti-Bacterial Agents*
  • Bacteremia / blood
  • Bacteremia / drug therapy*
  • Bacteremia / immunology
  • Drug Therapy, Combination / therapeutic use*
  • Endotoxins / blood*
  • Female
  • Gram-Negative Bacterial Infections / blood
  • Gram-Negative Bacterial Infections / drug therapy*
  • Gram-Negative Bacterial Infections / immunology
  • Humans
  • Interleukin-6 / blood
  • Interleukin-8 / blood
  • Male
  • Middle Aged
  • Shock, Septic / blood
  • Shock, Septic / drug therapy*
  • Shock, Septic / immunology
  • Tumor Necrosis Factor-alpha / metabolism

Substances

  • Anti-Bacterial Agents
  • Endotoxins
  • Interleukin-6
  • Interleukin-8
  • Tumor Necrosis Factor-alpha