[Precapillary pulmonary hypertension of uncertain etiology]

Dtsch Med Wochenschr. 1998 Jul 10;123(28-29):861-5. doi: 10.1055/s-2007-1024078.
[Article in German]

Abstract

History and clinical findings: A 39-year-old man was hospitalized for investigation of increasing dyspnoea for 3 month. On admission he was found to have bilateral ankle oedema, an enlarged liver and loud systolic murmur over the lower sternum.

Investigations: There were signs of right heart strain/hypertrophy on the chest radiogram and echocardiogram. After treatment of right heart failure cardiac catheterization indicated moderate precapillary pulmonary hypertension (PH) with a mean pulmonary artery pressure (PAPm) of 24 mm Hg and pulmonary vascular resistance (PVR) of 470 dyn.s.cm-5 at rest. All known causes having been excluded, the PH was classified as idiopathic.

Treatment and course: Evidence of acute pulmonary vascular reactivity was obtained with nitric oxide (NO) inhalation and oral diltiazem, a calcium-channel blocker. The latter, at a dosage of 3 x 120 mg daily, had after 13 days achieved a persisting reduction of PVR at rest and a reduction in PAP rise during exercise.

Conclusion: After exclusion of other causes, the acute right heart failure was found to be due to primary pulmonary hypertension. The therapeutic efficacity of diltiazem as a vasodilator can be predicted from the response to inhaled NO.

Publication types

  • Case Reports
  • English Abstract

MeSH terms

  • Adult
  • Calcium Channel Blockers / pharmacology
  • Calcium Channel Blockers / therapeutic use
  • Cardiac Catheterization
  • Diagnosis, Differential
  • Diltiazem / pharmacology
  • Diltiazem / therapeutic use
  • Dyspnea / etiology*
  • Heart Failure / complications
  • Heart Failure / etiology*
  • Humans
  • Hypertension, Pulmonary / complications*
  • Hypertension, Pulmonary / drug therapy
  • Hypertension, Pulmonary / etiology
  • Male
  • Nitric Oxide
  • Pulmonary Wedge Pressure / drug effects
  • Vascular Resistance / drug effects
  • Vasodilator Agents / pharmacology
  • Vasodilator Agents / therapeutic use
  • Ventricular Dysfunction, Right / etiology

Substances

  • Calcium Channel Blockers
  • Vasodilator Agents
  • Nitric Oxide
  • Diltiazem