CD8+ T-lymphocyte activation in HIV-1 disease reflects an aspect of pathogenesis distinct from viral burden and immunodeficiency

J Acquir Immune Defic Syndr Hum Retrovirol. 1998 Aug 1;18(4):332-40. doi: 10.1097/00042560-199808010-00004.

Abstract

The CD8+ T-cell response is central to control and eventual elimination of persistent viral infections. Although it might be expected that CD8+ T-cell activation would be associated with a better clinical outcome during viral infections, in long-term HIV-1 infection, high levels of CD8+ T-cell activation are instead associated with faster disease progression. In this study, cell surface expression of CD38, a flow cytometric marker of T-cell activation of CD8+ T cells, had predictive value for HIV-1 disease progression that was in part independent of the predictive value of plasma viral burden and CD4+ T-cell number. Measurements of CD38 antigen expression on CD8+ T cells in HIV-1-infected patients may be of value for assessing prognosis and the impact of therapeutic interventions. The pathogenetic reason why CD8+ T-cell activation is associated with poor outcome in HIV-1 disease remains unknown. Possibly CD8+ T-cell activation contributes to immunologic exhaustion, hyporesponsiveness of T cells to their cognate antigens, or perturbations in the T-cell receptor repertoire.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • ADP-ribosyl Cyclase
  • ADP-ribosyl Cyclase 1
  • Antigens, CD / analysis
  • Antigens, Differentiation / analysis
  • Antiviral Agents / therapeutic use
  • Biomarkers / analysis
  • CD4 Lymphocyte Count
  • CD4-Positive T-Lymphocytes / immunology
  • CD8-Positive T-Lymphocytes / immunology*
  • Chi-Square Distribution
  • Cohort Studies
  • Disease Progression
  • Disease-Free Survival
  • Follow-Up Studies
  • HIV Infections / drug therapy
  • HIV Infections / etiology*
  • HIV Infections / immunology
  • HIV Infections / virology
  • HIV-1* / genetics
  • HIV-1* / immunology
  • HIV-1* / physiology
  • Humans
  • Lymphocyte Activation*
  • Male
  • Membrane Glycoproteins
  • NAD+ Nucleosidase / analysis
  • Prognosis
  • Proportional Hazards Models
  • RNA, Viral / blood
  • Viral Load*

Substances

  • Antigens, CD
  • Antigens, Differentiation
  • Antiviral Agents
  • Biomarkers
  • Membrane Glycoproteins
  • RNA, Viral
  • ADP-ribosyl Cyclase
  • CD38 protein, human
  • NAD+ Nucleosidase
  • ADP-ribosyl Cyclase 1