Chemotherapy primes malignant glioma cells for CD95 ligand-induced apoptosis up-stream of caspase 3 activation

Eur J Pharmacol. 1998 Jul 3;352(1):111-5. doi: 10.1016/s0014-2999(98)00335-5.

Abstract

The cytotoxic cytokine, CD95 ligand, is an experimental anti-cancer agent. Here, we describe a novel pathway of drug-mediated augmentation of CD95 ligand-induced apoptosis. We report that prolonged pre-exposure of human malignant glioma cells to different cytotoxic agents, VM26, cytarabine and cisplatin, induces strong sensitization to CD95 ligand-induced apoptosis. CD95 gene transfer does not prevent sensitization, suggesting that sensitization is not mediated by drug-induced CD95 expression. Priming with cytotoxic drugs greatly increases CD95 ligand-induced caspase 3 activity, indicating that the cytotoxic drugs positively modulate the CD95-dependent signalling cascade up-stream of caspase 3 activation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antineoplastic Agents / pharmacology*
  • Brain Neoplasms / enzymology
  • Brain Neoplasms / pathology*
  • Caspase 3
  • Caspases / metabolism*
  • Enzyme Activation
  • Glioma / enzymology
  • Glioma / pathology*
  • Humans
  • Signal Transduction
  • Tumor Cells, Cultured
  • fas Receptor / metabolism*

Substances

  • Antineoplastic Agents
  • fas Receptor
  • CASP3 protein, human
  • Caspase 3
  • Caspases