Mapping an endometrial cancer tumor suppressor gene at 10q25 and development of a bacterial clone contig for the consensus deletion interval

Genomics. 1998 Aug 15;52(1):9-16. doi: 10.1006/geno.1998.5399.

Abstract

Frequent loss of chromosome 10q sequences in endometrial cancers suggests the involvement of a tumor suppressor gene. Previous loss-of-heterozygosity (LOH)studies have pointed to the 10q25-q26 region as the likely site of a tumor suppressor involved in endometrial tumorigenesis (S. L. Peiffer et al., 1995, Cancer Res. 55: 1922-1926; S. Nagase et al., 1996, Br. J. Cancer 74: 1979-1983; S. Nagase et al.,1997, Cancer Res. 57: 1630-1633). In an attempt to define further the localization of a tumor suppressor gene at 10q25, we screened a panel of 123 endometrioid adenocarcinomas for loss of heterozygosity of 10q25.3 sequences. Forty-three (35%) revealed LOH at one or more loci. The observed patterns of allelic loss define a minimum consensus region of deletion between D10S221 and D10S610. A sequence-ready bacterial clone contig and a long-range restriction map for a 1-Mb interval spanning the deletion region were developed as the first step in experiments directed toward the discovery the 10q25 tumor suppressor.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Carcinoma, Endometrioid / genetics*
  • Chromosome Mapping* / methods
  • Chromosomes, Artificial, Yeast / genetics
  • Chromosomes, Bacterial / genetics
  • Chromosomes, Human, Pair 10 / genetics*
  • Cloning, Molecular
  • Female
  • Genes, Tumor Suppressor / genetics*
  • Genetic Markers / genetics
  • Humans
  • Loss of Heterozygosity / genetics
  • Restriction Mapping
  • Sequence Deletion / genetics

Substances

  • Genetic Markers