Angiogenesis in P- and E-selectin-deficient mice

Microcirculation. 1998;5(2-3):173-8.

Abstract

Objectives: Several observations reported earlier indicated that the selectins, in particular E-selectin, might be involved in angiogenesis; however, mice deficient in the endothelial selectins develop normally. To clarify the role of endothelial selectins in angiogenesis, we have studied experimentally induced angiogenesis in selectin-deficient mice.

Methods: Hydron pellets containing either basic fibroblast growth factor or the inflammatory cytokine tumor necrosis factor alpha were implanted into the corneas of wild-type and P- and/or E-selectin-deficient mice.

Results: The lengths and circumferential range of the newly formed blood vessels in the corneas of the endothelial selectin-deficient mice were similar to those of wild-type mice.

Conclusion: The endothelial selectins are not essential in experimentally induced angiogenesis in vivo.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Cornea / blood supply
  • Cornea / drug effects
  • E-Selectin / genetics
  • E-Selectin / physiology*
  • Fibroblast Growth Factor 2 / administration & dosage
  • Fibroblast Growth Factor 2 / pharmacology
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Neovascularization, Physiologic* / drug effects
  • P-Selectin / genetics
  • P-Selectin / physiology*
  • Recombinant Proteins / administration & dosage
  • Recombinant Proteins / pharmacology
  • Tumor Necrosis Factor-alpha / administration & dosage
  • Tumor Necrosis Factor-alpha / pharmacology

Substances

  • E-Selectin
  • P-Selectin
  • Recombinant Proteins
  • Tumor Necrosis Factor-alpha
  • Fibroblast Growth Factor 2