Quantitative differences in the active-site hydrophobicity of five human glutathione S-transferase isoenzymes: water-soluble carcinogen-selective properties of the neoplastic GSTP1-1 species

Arch Biochem Biophys. 1999 Jan 15;361(2):271-6. doi: 10.1006/abbi.1998.0983.

Abstract

The active-site (the H-site) hydrophobicity of five human glutathione S-transferases (GSTs) was analyzed by application of linear free energy relationships (LFERs) with a series of S-alkylated glutathione inhibitors, GS(CH2)n - 1CH3 (n = 1-14). Distinct linear reltionships were observed in the plots of log Ki (inhibition constant) vs n for the five forms, whereby the Kis varied by three to four orders of magnitude. Mean free enthalpy changes per methylene group (-Delta DeltaG degreess), a measure of H-site hydrophobicity, were in the order M1-1 (4.6 kJ/mol) > A1-1 (3. 9 kJ/mol) > A1-2 (3.8 kJ/mol) > A2-2 (2.8 kJ/mol) > P1-1 (1.6 kJ/mol). The quantitative differences may in part account for the extraordinary broad and overlapping substrate specificities of the Alpha-, Mu-, and Pi-class isoenzymes. In contrast to the Alpha and Mu classes being selective for strongly electrophilic compounds, the neoplastic P1-1 species was indicated to be selective for weakly electrophilic and water-soluble carcinogens such as acrolein and hydroxyalkenals.

MeSH terms

  • Binding Sites
  • Carcinogens / chemistry*
  • Carcinogens / metabolism
  • Enzyme Activation / drug effects
  • Enzyme Activation / physiology
  • Enzyme Inhibitors / pharmacology
  • Glutathione / metabolism
  • Glutathione S-Transferase pi
  • Glutathione Transferase / antagonists & inhibitors
  • Glutathione Transferase / chemistry*
  • Glutathione Transferase / metabolism
  • Humans
  • Isoenzymes / antagonists & inhibitors
  • Isoenzymes / chemistry*
  • Isoenzymes / metabolism
  • Neoplasm Proteins / antagonists & inhibitors
  • Neoplasm Proteins / chemistry*
  • Neoplasm Proteins / metabolism
  • Protein Binding / drug effects
  • Solubility
  • Thermodynamics
  • Water / metabolism*

Substances

  • Carcinogens
  • Enzyme Inhibitors
  • Isoenzymes
  • Neoplasm Proteins
  • Water
  • GSTP1 protein, human
  • Glutathione S-Transferase pi
  • Glutathione Transferase
  • Glutathione