Differential expression of an orphan receptor COUP-TFI and corepressors in adrenal tumors

Endocr Res. 1998 Aug-Nov;24(3-4):881-5. doi: 10.3109/07435809809032703.

Abstract

Recently, it has been shown that CYP17 gene transcription is activated by SF-1 (Steroidogenic Factor-1) binding to a cyclic AMP-responsive sequence within the promoter region of the gene, whereas it is inhibited by COUP-TF (Chicken Ovalbumin Upstream Promoter-Transcription Factor) binding to the sequence. We have shown that transcriptional repression by COUP-TFI is mediated by corepressors, N-CoR (nuclear receptor corepressor) and SMRT (silencing mediator for retinoid and thyroid hormone-receptor). Therefore, we compared the expression of COUP-TFI, N-CoR and SMRT in non-hyperfunctioning adrenocortical adenomas and normal adrenal glands. We found significantly higher expression of COUP-TFI mRNA in non-hyperfunctioning adenomas (n=5, 227+/-18%) than in normal adrenals (n=5, 96+/-4%). Interestingly, the pattern of N-CoR and SMRT expression was different compared with COUP-TFI expression. These data suggest that COUP-TFI, N-CoR, and SMRT may play a differential role in steroid biosynthesis of non-hyperfunctioning adenomas.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenoma / metabolism*
  • Adrenal Gland Neoplasms / metabolism*
  • Adrenal Glands / metabolism
  • Adult
  • Aged
  • COUP Transcription Factors
  • DNA-Binding Proteins / metabolism*
  • Female
  • Humans
  • Male
  • Middle Aged
  • Nuclear Proteins / metabolism
  • Nuclear Receptor Co-Repressor 1
  • Nuclear Receptor Co-Repressor 2
  • Receptors, Steroid*
  • Reference Values
  • Repressor Proteins / metabolism*
  • Steroidogenic Factor 1
  • Transcription Factors / metabolism*

Substances

  • COUP Transcription Factors
  • DNA-Binding Proteins
  • NCOR1 protein, human
  • NCOR2 protein, human
  • NR5A1 protein, human
  • Nuclear Proteins
  • Nuclear Receptor Co-Repressor 1
  • Nuclear Receptor Co-Repressor 2
  • Receptors, Steroid
  • Repressor Proteins
  • Steroidogenic Factor 1
  • Transcription Factors