Samson LD - Search Results

1

DNA repair methyltransferase (Mgmt) knockout mice are sensitive to the lethal effects of chemotherapeutic alkylating agents.

Glassner BJ, Weeda G, Allan JM, Broekhof JL, Carls NH, Donker I, Engelward BP, Hampson RJ, Hersmus R, Hickman MJ, Roth RB, Warren HB, Wu MM, Hoeijmakers JH, Samson LD. Glassner BJ, et al. Among authors: samson ld. Mutagenesis. 1999 May;14(3):339-47. doi: 10.1093/mutage/14.3.339. Mutagenesis. 1999. PMID: 10375003

2

Role of DNA mismatch repair and p53 in signaling induction of apoptosis by alkylating agents.

Hickman MJ, Samson LD. Hickman MJ, et al. Among authors: samson ld. Proc Natl Acad Sci U S A. 1999 Sep 14;96(19):10764-9. doi: 10.1073/pnas.96.19.10764. Proc Natl Acad Sci U S A. 1999. PMID: 10485900 Free PMC article.

3

Gene transfer to suppress bone marrow alkylation sensitivity.

Roth RB, Samson LD. Roth RB, et al. Among authors: samson ld. Mutat Res. 2000 Apr;462(2-3):107-20. doi: 10.1016/s1383-5742(00)00021-1. Mutat Res. 2000. PMID: 10767622 Review.

4

Base excision repair deficient mice lacking the Aag alkyladenine DNA glycosylase.

Engelward BP, Weeda G, Wyatt MD, Broekhof JL, de Wit J, Donker I, Allan JM, Gold B, Hoeijmakers JH, Samson LD. Engelward BP, et al. Among authors: samson ld. Proc Natl Acad Sci U S A. 1997 Nov 25;94(24):13087-92. doi: 10.1073/pnas.94.24.13087. Proc Natl Acad Sci U S A. 1997. PMID: 9371804 Free PMC article.

5

Mammalian 3-methyladenine DNA glycosylase protects against the toxicity and clastogenicity of certain chemotherapeutic DNA cross-linking agents.

Allan JM, Engelward BP, Dreslin AJ, Wyatt MD, Tomasz M, Samson LD. Allan JM, et al. Among authors: samson ld. Cancer Res. 1998 Sep 1;58(17):3965-73. Cancer Res. 1998. PMID: 9731510

6

3-Methyladenine DNA glycosylase-deficient Aag null mice display unexpected bone marrow alkylation resistance.

Roth RB, Samson LD. Roth RB, et al. Among authors: samson ld. Cancer Res. 2002 Feb 1;62(3):656-60. Cancer Res. 2002. PMID: 11830515

7

A chemical and genetic approach together define the biological consequences of 3-methyladenine lesions in the mammalian genome.

Engelward BP, Allan JM, Dreslin AJ, Kelly JD, Wu MM, Gold B, Samson LD. Engelward BP, et al. Among authors: samson ld. J Biol Chem. 1998 Feb 27;273(9):5412-8. doi: 10.1074/jbc.273.9.5412. J Biol Chem. 1998. PMID: 9479003 Free article.

8

Increasing DNA repair methyltransferase levels via bone marrow stem cell transduction rescues mice from the toxic effects of 1,3-bis(2-chloroethyl)-1-nitrosourea, a chemotherapeutic alkylating agent.

Maze R, Carney JP, Kelley MR, Glassner BJ, Williams DA, Samson L. Maze R, et al. Proc Natl Acad Sci U S A. 1996 Jan 9;93(1):206-10. doi: 10.1073/pnas.93.1.206. Proc Natl Acad Sci U S A. 1996. PMID: 8552605 Free PMC article.

9

Repair-deficient 3-methyladenine DNA glycosylase homozygous mutant mouse cells have increased sensitivity to alkylation-induced chromosome damage and cell killing.

Engelward BP, Dreslin A, Christensen J, Huszar D, Kurahara C, Samson L. Engelward BP, et al. EMBO J. 1996 Feb 15;15(4):945-52. EMBO J. 1996. PMID: 8631315 Free PMC article.

10

The influence of DNA glycosylases on spontaneous mutation.

Glassner BJ, Posnick LM, Samson LD. Glassner BJ, et al. Among authors: samson ld. Mutat Res. 1998 May 25;400(1-2):33-44. doi: 10.1016/s0027-5107(98)00045-1. Mutat Res. 1998. PMID: 9685578 Review. No abstract available.

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