Balmanno K - Search Results

1

Regulation of cell cycle re-entry by growth, survival and stress signalling pathways.

Cook SJ, Balmanno K, Garner A, Millar T, Taverner C, Todd D. Cook SJ, et al. Among authors: balmanno k. Biochem Soc Trans. 2000 Feb;28(2):233-40. doi: 10.1042/bst0280233. Biochem Soc Trans. 2000. PMID: 10816134 Review.

2

Sustained MAP kinase activation is required for the expression of cyclin D1, p21Cip1 and a subset of AP-1 proteins in CCL39 cells.

Balmanno K, Cook SJ. Balmanno K, et al. Oncogene. 1999 May 20;18(20):3085-97. doi: 10.1038/sj.onc.1202647. Oncogene. 1999. PMID: 10340380

3

Tumour cell survival signalling by the ERK1/2 pathway.

Balmanno K, Cook SJ. Balmanno K, et al. Cell Death Differ. 2009 Mar;16(3):368-77. doi: 10.1038/cdd.2008.148. Epub 2008 Oct 10. Cell Death Differ. 2009. PMID: 18846109 Review.

4

ERK1/2 and p38 cooperate to delay progression through G1 by promoting cyclin D1 protein turnover.

Densham RM, Todd DE, Balmanno K, Cook SJ. Densham RM, et al. Among authors: balmanno k. Cell Signal. 2008 Nov;20(11):1986-94. doi: 10.1016/j.cellsig.2008.07.005. Epub 2008 Jul 10. Cell Signal. 2008. PMID: 18664382

5

Colorectal cancer cells with the BRAF(V600E) mutation are addicted to the ERK1/2 pathway for growth factor-independent survival and repression of BIM.

Wickenden JA, Jin H, Johnson M, Gillings AS, Newson C, Austin M, Chell SD, Balmanno K, Pritchard CA, Cook SJ. Wickenden JA, et al. Among authors: balmanno k. Oncogene. 2008 Dec 4;27(57):7150-61. doi: 10.1038/onc.2008.335. Epub 2008 Sep 22. Oncogene. 2008. PMID: 18806830 Free PMC article.

6

Tumour cell responses to new fibroblast growth factor receptor tyrosine kinase inhibitors and identification of a gatekeeper mutation in FGFR3 as a mechanism of acquired resistance.

Chell V, Balmanno K, Little AS, Wilson M, Andrews S, Blockley L, Hampson M, Gavine PR, Cook SJ. Chell V, et al. Among authors: balmanno k. Oncogene. 2013 Jun 20;32(25):3059-70. doi: 10.1038/onc.2012.319. Epub 2012 Aug 6. Oncogene. 2013. PMID: 22869148

7

ERK1/2 inhibitors act as monovalent degraders inducing ubiquitylation and proteasome-dependent turnover of ERK2, but not ERK1.

Balmanno K, Kidger AM, Byrne DP, Sale MJ, Nassman N, Eyers PA, Cook SJ. Balmanno K, et al. Biochem J. 2023 May 15;480(9):587-605. doi: 10.1042/BCJ20220598. Biochem J. 2023. PMID: 37018014 Free PMC article.

8

IKKα plays a major role in canonical NF-κB signalling in colorectal cells.

Prescott JA, Balmanno K, Mitchell JP, Okkenhaug H, Cook SJ. Prescott JA, et al. Among authors: balmanno k. Biochem J. 2022 Feb 11;479(3):305-325. doi: 10.1042/BCJ20210783. Biochem J. 2022. PMID: 35029639 Free PMC article.

9

Dual-Mechanism ERK1/2 Inhibitors Exploit a Distinct Binding Mode to Block Phosphorylation and Nuclear Accumulation of ERK1/2.

Kidger AM, Munck JM, Saini HK, Balmanno K, Minihane E, Courtin A, Graham B, O'Reilly M, Odle R, Cook SJ. Kidger AM, et al. Among authors: balmanno k. Mol Cancer Ther. 2020 Feb;19(2):525-539. doi: 10.1158/1535-7163.MCT-19-0505. Epub 2019 Nov 20. Mol Cancer Ther. 2020. PMID: 31748345

10

An mTORC1-to-CDK1 Switch Maintains Autophagy Suppression during Mitosis.

Odle RI, Walker SA, Oxley D, Kidger AM, Balmanno K, Gilley R, Okkenhaug H, Florey O, Ktistakis NT, Cook SJ. Odle RI, et al. Among authors: balmanno k. Mol Cell. 2020 Jan 16;77(2):228-240.e7. doi: 10.1016/j.molcel.2019.10.016. Epub 2019 Nov 13. Mol Cell. 2020. PMID: 31733992 Free PMC article.

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