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Investigation of a truncated cardiac troponin T that causes familial hypertrophic cardiomyopathy: Ca(2+) regulatory properties of reconstituted thin filaments depend on the ratio of mutant to wild-type protein.
Circ Res. 2000 Jun 9;86(11):1146-52. doi: 10.1161/01.res.86.11.1146.
Circ Res. 2000.
PMID: 10850966
Effects of two hypertrophic cardiomyopathy mutations in alpha-tropomyosin, Asp175Asn and Glu180Gly, on Ca2+ regulation of thin filament motility.
Bing W, Redwood CS, Purcell IF, Esposito G, Watkins H, Marston SB.
Bing W, et al.
Biochem Biophys Res Commun. 1997 Jul 30;236(3):760-4. doi: 10.1006/bbrc.1997.7045.
Biochem Biophys Res Commun. 1997.
PMID: 9245729
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Effect of hypertrophic cardiomyopathy mutations in human cardiac muscle alpha -tropomyosin (Asp175Asn and Glu180Gly) on the regulatory properties of human cardiac troponin determined by in vitro motility assay.
Bing W, Knott A, Redwood C, Esposito G, Purcell I, Watkins H, Marston S.
Bing W, et al.
J Mol Cell Cardiol. 2000 Aug;32(8):1489-98. doi: 10.1006/jmcc.2000.1182.
J Mol Cell Cardiol. 2000.
PMID: 10900175
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Peripheral entrapment neuropathies of upper extremity.
Esposito GM.
Esposito GM.
N Y State J Med. 1972 Mar 15;72(6):717-24.
N Y State J Med. 1972.
PMID: 4334934
No abstract available.
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