Khoo C - Search Results

1

Modeling chromosomal instability and epithelial carcinogenesis in the telomerase-deficient mouse.

Chang S, Khoo C, DePinho RA. Chang S, et al. Among authors: khoo c. Semin Cancer Biol. 2001 Jun;11(3):227-39. doi: 10.1006/scbi.2000.0374. Semin Cancer Biol. 2001. PMID: 11407947 Review.

2

Telomere-based crisis: functional differences between telomerase activation and ALT in tumor progression.

Chang S, Khoo CM, Naylor ML, Maser RS, DePinho RA. Chang S, et al. Genes Dev. 2003 Jan 1;17(1):88-100. doi: 10.1101/gad.1029903. Genes Dev. 2003. PMID: 12514102 Free PMC article.

3

Ink4a/Arf tumor suppressor does not modulate the degenerative conditions or tumor spectrum of the telomerase-deficient mouse.

Khoo CM, Carrasco DR, Bosenberg MW, Paik JH, Depinho RA. Khoo CM, et al. Proc Natl Acad Sci U S A. 2007 Mar 6;104(10):3931-6. doi: 10.1073/pnas.0700093104. Epub 2007 Feb 27. Proc Natl Acad Sci U S A. 2007. PMID: 17360455 Free PMC article.

4

Telomeric protein Pin2/TRF1 as an important ATM target in response to double strand DNA breaks.

Kishi S, Zhou XZ, Ziv Y, Khoo C, Hill DE, Shiloh Y, Lu KP. Kishi S, et al. Among authors: khoo c. J Biol Chem. 2001 Aug 3;276(31):29282-91. doi: 10.1074/jbc.M011534200. Epub 2001 May 25. J Biol Chem. 2001. PMID: 11375976 Free article.

5

Effects of estrogenic oral contraceptives on the lipoprotein B particle system defined by apolipoproteins E and C-III content.

Khoo C, Campos H, Judge H, Sacks FM. Khoo C, et al. J Lipid Res. 1999 Feb;40(2):202-12. J Lipid Res. 1999. PMID: 9925648 Free article.

Fasting plasma was separated by anti-apoE and C-III affinity chromatography and ultracentrifugation into four types of VLDL, IDL, and LDL particles: with apoE but not apoC-III (E+C-), apoC-III but not apoE (E-C+), both (E+C+) or neither (E-C-). …

Fasting plasma was separated by anti-apoE and C-III affinity chromatography and ultracentrifugation into four types of VLDL, IDL, and …

6

Rapid turnover of apolipoprotein C-III-containing triglyceride-rich lipoproteins contributing to the formation of LDL subfractions.

Zheng C, Khoo C, Ikewaki K, Sacks FM. Zheng C, et al. Among authors: khoo c. J Lipid Res. 2007 May;48(5):1190-203. doi: 10.1194/jlr.P600011-JLR200. Epub 2007 Feb 21. J Lipid Res. 2007. PMID: 17314277 Free article.

7

Diurnal and acute patterns of postprandial apolipoprotein B-48 in VLDL, IDL, and LDL from normolipidemic humans.

Campos H, Khoo C, Sacks FM. Campos H, et al. Among authors: khoo c. Atherosclerosis. 2005 Aug;181(2):345-51. doi: 10.1016/j.atherosclerosis.2004.12.045. Epub 2005 Feb 19. Atherosclerosis. 2005. PMID: 16039289

8

Apolipoprotein C-III and the metabolic basis for hypertriglyceridemia and the dense low-density lipoprotein phenotype.

Zheng C, Khoo C, Furtado J, Sacks FM. Zheng C, et al. Among authors: khoo c. Circulation. 2010 Apr 20;121(15):1722-34. doi: 10.1161/CIRCULATIONAHA.109.875807. Epub 2010 Apr 5. Circulation. 2010. PMID: 20368524 Free PMC article.

9

Distinct patterns of lipoproteins with apoB defined by presence of apoE or apoC-III in hypercholesterolemia and hypertriglyceridemia.

Campos H, Perlov D, Khoo C, Sacks FM. Campos H, et al. Among authors: khoo c. J Lipid Res. 2001 Aug;42(8):1239-49. J Lipid Res. 2001. PMID: 11483625 Free article.

10

Dietary monounsaturated fat activates metabolic pathways for triglyceride-rich lipoproteins that involve apolipoproteins E and C-III.

Zheng C, Khoo C, Furtado J, Ikewaki K, Sacks FM. Zheng C, et al. Among authors: khoo c. Am J Clin Nutr. 2008 Aug;88(2):272-81. doi: 10.1093/ajcn/88.2.272. Am J Clin Nutr. 2008. PMID: 18689361 Free PMC article. Clinical Trial.

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