Abraham E - Search Results

1

Extracellular superoxide dismutase attenuates lung injury after hemorrhage.

Bowler RP, Arcaroli J, Crapo JD, Ross A, Slot JW, Abraham E. Bowler RP, et al. Among authors: abraham e. Am J Respir Crit Care Med. 2001 Jul 15;164(2):290-4. doi: 10.1164/ajrccm.164.2.2011054. Am J Respir Crit Care Med. 2001. PMID: 11463603

2

Evidence for extracellular superoxide dismutase as a mediator of hemorrhage-induced lung injury.

Bowler RP, Arcaroli J, Abraham E, Patel M, Chang LY, Crapo JD. Bowler RP, et al. Among authors: abraham e. Am J Physiol Lung Cell Mol Physiol. 2003 Apr;284(4):L680-7. doi: 10.1152/ajplung.00191.2002. Epub 2003 Jan 10. Am J Physiol Lung Cell Mol Physiol. 2003. PMID: 12618426 Free article.

3

Early alterations in neutrophil activation are associated with outcome in acute lung injury.

Yang KY, Arcaroli JJ, Abraham E. Yang KY, et al. Among authors: abraham e. Am J Respir Crit Care Med. 2003 Jun 1;167(11):1567-74. doi: 10.1164/rccm.200207-664OC. Epub 2003 Mar 5. Am J Respir Crit Care Med. 2003. PMID: 12626346

4

Extracellular superoxide dismutase haplotypes are associated with acute lung injury and mortality.

Arcaroli JJ, Hokanson JE, Abraham E, Geraci M, Murphy JR, Bowler RP, Dinarello CA, Silveira L, Sankoff J, Heyland D, Wischmeyer P, Crapo JD. Arcaroli JJ, et al. Among authors: abraham e. Am J Respir Crit Care Med. 2009 Jan 15;179(2):105-12. doi: 10.1164/rccm.200710-1566OC. Epub 2008 Oct 23. Am J Respir Crit Care Med. 2009. PMID: 18948423 Free PMC article.

5

Pulmonary edema fluid antioxidants are depressed in acute lung injury.

Bowler RP, Velsor LW, Duda B, Chan ED, Abraham E, Ware LB, Matthay MA, Day BJ. Bowler RP, et al. Among authors: abraham e. Crit Care Med. 2003 Sep;31(9):2309-15. doi: 10.1097/01.CCM.0000085090.06078.8C. Crit Care Med. 2003. PMID: 14501961

6

Hemorrhage-induced acute lung injury is TLR-4 dependent.

Barsness KA, Arcaroli J, Harken AH, Abraham E, Banerjee A, Reznikov L, McIntyre RC. Barsness KA, et al. Among authors: abraham e. Am J Physiol Regul Integr Comp Physiol. 2004 Sep;287(3):R592-9. doi: 10.1152/ajpregu.00412.2003. Epub 2004 Apr 8. Am J Physiol Regul Integr Comp Physiol. 2004. PMID: 15072965 Free article.

7

Neutrophils as early immunologic effectors in hemorrhage- or endotoxemia-induced acute lung injury.

Abraham E, Carmody A, Shenkar R, Arcaroli J. Abraham E, et al. Am J Physiol Lung Cell Mol Physiol. 2000 Dec;279(6):L1137-45. doi: 10.1152/ajplung.2000.279.6.L1137. Am J Physiol Lung Cell Mol Physiol. 2000. PMID: 11076804 Free article.

8

Activation of extracellular signal-regulated kinases, NF-kappa B, and cyclic adenosine 5'-monophosphate response element-binding protein in lung neutrophils occurs by differing mechanisms after hemorrhage or endotoxemia.

Abraham E, Arcaroli J, Shenkar R. Abraham E, et al. J Immunol. 2001 Jan 1;166(1):522-30. doi: 10.4049/jimmunol.166.1.522. J Immunol. 2001. PMID: 11123332

9

Interactions between CBP, NF-kappaB, and CREB in the lungs after hemorrhage and endotoxemia.

Shenkar R, Yum HK, Arcaroli J, Kupfner J, Abraham E. Shenkar R, et al. Among authors: abraham e. Am J Physiol Lung Cell Mol Physiol. 2001 Aug;281(2):L418-26. doi: 10.1152/ajplung.2001.281.2.L418. Am J Physiol Lung Cell Mol Physiol. 2001. PMID: 11435217 Free article.

10

Role of p38 MAP kinase in the development of acute lung injury.

Arcaroli J, Yum HK, Kupfner J, Park JS, Yang KY, Abraham E. Arcaroli J, et al. Among authors: abraham e. Clin Immunol. 2001 Nov;101(2):211-9. doi: 10.1006/clim.2001.5108. Clin Immunol. 2001. PMID: 11683580

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