Yamasaki H - Search Results

1

Reduction of malignant phenotype of HEPG2 cell is associated with the expression of connexin 26 but not connexin 32.

Yano T, Hernandez-Blazquez FJ, Omori Y, Yamasaki H. Yano T, et al. Among authors: yamasaki h. Carcinogenesis. 2001 Oct;22(10):1593-600. doi: 10.1093/carcin/22.10.1593. Carcinogenesis. 2001. PMID: 11576997

2

Delayed liver regeneration and increased susceptibility to chemical hepatocarcinogenesis in transgenic mice expressing a dominant-negative mutant of connexin32 only in the liver.

Dagli ML, Yamasaki H, Krutovskikh V, Omori Y. Dagli ML, et al. Among authors: yamasaki h. Carcinogenesis. 2004 Apr;25(4):483-92. doi: 10.1093/carcin/bgh050. Epub 2003 Dec 19. Carcinogenesis. 2004. PMID: 14688024

3

Connexin genes and cell growth control.

Yamasaki H, Krutovskikh V, Mesnil M, Omori Y. Yamasaki H, et al. Arch Toxicol Suppl. 1996;18:105-14. doi: 10.1007/978-3-642-61105-6_12. Arch Toxicol Suppl. 1996. PMID: 8678786 Review. No abstract available.

4

Connexin 32 mutations from X-linked Charcot-Marie-Tooth disease patients: functional defects and dominant negative effects.

Omori Y, Mesnil M, Yamasaki H. Omori Y, et al. Among authors: yamasaki h. Mol Biol Cell. 1996 Jun;7(6):907-16. doi: 10.1091/mbc.7.6.907. Mol Biol Cell. 1996. PMID: 8816997 Free PMC article.

5

Cx32 gene mutation in a chemically induced rat liver tumour.

Omori Y, Krutovskikh V, Mironov N, Tsuda H, Yamasaki H. Omori Y, et al. Among authors: yamasaki h. Carcinogenesis. 1996 Sep;17(9):2077-80. doi: 10.1093/carcin/17.9.2077. Carcinogenesis. 1996. PMID: 8824538

6

Mutated connexin43 proteins inhibit rat glioma cell growth suppression mediated by wild-type connexin43 in a dominant-negative manner.

Omori Y, Yamasaki H. Omori Y, et al. Among authors: yamasaki h. Int J Cancer. 1998 Nov 9;78(4):446-53. doi: 10.1002/(sici)1097-0215(19981109)78:4<446::aid-ijc10>3.0.co;2-4. Int J Cancer. 1998. PMID: 9797133

7

Growth control of 3T3 fibroblast cell lines established from connexin 43-deficient mice.

Yamakage K, Omori Y, Piccoli C, Yamasaki H. Yamakage K, et al. Among authors: yamasaki h. Mol Carcinog. 1998 Oct;23(2):121-8. doi: 10.1002/(sici)1098-2744(199810)23:2<121::aid-mc9>3.0.co;2-p. Mol Carcinog. 1998. PMID: 9808166

8

Role of connexin (gap junction) genes in cell growth control: approach with site-directed mutagenesis and dominant-negative effects.

Omori Y, Duflot-Dancer A, Mesnil M, Yamasaki H. Omori Y, et al. Among authors: yamasaki h. Toxicol Lett. 1998 Aug;96-97:105-10. doi: 10.1016/s0378-4274(98)00056-3. Toxicol Lett. 1998. PMID: 9820654

9

Role of connexin (gap junction) genes in cell growth control and carcinogenesis.

Yamasaki H, Krutovskikh V, Mesnil M, Tanaka T, Zaidan-Dagli ML, Omori Y. Yamasaki H, et al. C R Acad Sci III. 1999 Feb-Mar;322(2-3):151-9. doi: 10.1016/s0764-4469(99)80038-9. C R Acad Sci III. 1999. PMID: 10196667 Review.

10

Gap junction proteins connexin32 and connexin43 partially acquire growth-suppressive function in HeLa cells by deletion of their C-terminal tails.

Omori Y, Yamasaki H. Omori Y, et al. Among authors: yamasaki h. Carcinogenesis. 1999 Oct;20(10):1913-8. doi: 10.1093/carcin/20.10.1913. Carcinogenesis. 1999. PMID: 10506104

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