Jiang Y - Search Results

1

New approach to treatment of shock--restitution of vasoreactivity.

Zhao KS, Huang X, Liu J, Huang Q, Jin C, Jiang Y, Jin J, Zhao G. Zhao KS, et al. Among authors: jiang y. Shock. 2002 Aug;18(2):189-92. doi: 10.1097/00024382-200208000-00017. Shock. 2002. PMID: 12166785

2

Role of p38 MAPK in ICAM-1 expression of vascular endothelial cells induced by lipopolysaccharide.

Yan W, Zhao K, Jiang Y, Huang Q, Wang J, Kan W, Wang S. Yan W, et al. Among authors: jiang y. Shock. 2002 May;17(5):433-8. doi: 10.1097/00024382-200205000-00016. Shock. 2002. PMID: 12022767

3

Lung, spleen, and kidney are the major places for inducible nitric oxide synthase expression in endotoxic shock: role of p38 mitogen-activated protein kinase in signal transduction of inducible nitric oxide synthase expression.

Kan W, Zhao KS, Jiang Y, Yan W, Huang Q, Wang J, Qin Q, Huang X, Wang S. Kan W, et al. Among authors: jiang y. Shock. 2004 Mar;21(3):281-7. doi: 10.1097/01.shk.0000113314.37747.55. Shock. 2004. PMID: 14770043

4

p38 MAPK Signal is Necessary for TNF-alpha Gene Expression in RAW Cells.

Jiang Y, Liu AH, Huang QB. Jiang Y, et al. Sheng Wu Hua Xue Yu Sheng Wu Wu Li Xue Bao (Shanghai). 1999;31(1):9-15. Sheng Wu Hua Xue Yu Sheng Wu Wu Li Xue Bao (Shanghai). 1999. PMID: 12142907

5

Studies on the flow and distribution of leukocytes in mesentery microcirculation of rats.

Jiang Y, Liu AH, Zhao KS. Jiang Y, et al. World J Gastroenterol. 1999 Jun;5(3):231-234. doi: 10.3748/wjg.v5.i3.231. World J Gastroenterol. 1999. PMID: 11819437 Free PMC article.

6

Functional characterization of S100A8 and S100A9 in altering monolayer permeability of human umbilical endothelial cells.

Wang L, Luo H, Chen X, Jiang Y, Huang Q. Wang L, et al. Among authors: jiang y. PLoS One. 2014 Mar 3;9(3):e90472. doi: 10.1371/journal.pone.0090472. eCollection 2014. PLoS One. 2014. PMID: 24595267 Free PMC article.

7

Role of TLR4-p38 MAPK-Hsp27 signal pathway in LPS-induced pulmonary epithelial hyperpermeability.

Wang W, Weng J, Yu L, Huang Q, Jiang Y, Guo X. Wang W, et al. Among authors: jiang y. BMC Pulm Med. 2018 Nov 27;18(1):178. doi: 10.1186/s12890-018-0735-0. BMC Pulm Med. 2018. PMID: 30482200 Free PMC article.

8

Co-expression of KCNE2 and KChIP2c modulates the electrophysiological properties of Kv4.2 current in COS-7 cells.

Liu WJ, Wang HT, Chen WW, Deng JX, Jiang Y, Liu J. Liu WJ, et al. Among authors: jiang y. Acta Pharmacol Sin. 2008 Jun;29(6):653-60. doi: 10.1111/j.1745-7254.2008.00804.x. Acta Pharmacol Sin. 2008. PMID: 18501111

9

High-mobility group box 1 (HMGB1) downregulates cardiac transient outward potassium current (Ito) through downregulation of Kv4.2 and Kv4.3 channel transcripts and proteins.

Liu W, Deng J, Xu J, Wang H, Yuan M, Liu N, Jiang Y, Liu J. Liu W, et al. Among authors: jiang y. J Mol Cell Cardiol. 2010 Sep;49(3):438-48. doi: 10.1016/j.yjmcc.2010.05.004. Epub 2010 May 17. J Mol Cell Cardiol. 2010. PMID: 20483361

10

β-Catenin phosphorylation at Y654 and Y142 is crucial for high mobility group box-1 protein-induced pulmonary vascular hyperpermeability.

Weng J, Yu L, Chen Z, Su H, Yu S, Zhang Y, Lei X, Chen L, Cui Y, Huang Q, Jiang Y, Guo X. Weng J, et al. Among authors: jiang y. J Mol Cell Cardiol. 2019 Feb;127:174-184. doi: 10.1016/j.yjmcc.2018.12.012. Epub 2018 Dec 25. J Mol Cell Cardiol. 2019. PMID: 30592964

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