Klein J - Search Results

1

Isoproterenol is a positive regulator of the suppressor of cytokine signaling-3 gene expression in 3T3-L1 adipocytes.

Fasshauer M, Klein J, Lossner U, Paschke R. Fasshauer M, et al. Among authors: klein j. J Endocrinol. 2002 Dec;175(3):727-33. doi: 10.1677/joe.0.1750727. J Endocrinol. 2002. PMID: 12475383

2

Isoproterenol inhibits resistin gene expression through a G(S)-protein-coupled pathway in 3T3-L1 adipocytes.

Fasshauer M, Klein J, Neumann S, Eszlinger M, Paschke R. Fasshauer M, et al. Among authors: klein j. FEBS Lett. 2001 Jun 29;500(1-2):60-3. doi: 10.1016/s0014-5793(01)02588-1. FEBS Lett. 2001. PMID: 11434927 Free article.

3

Adiponectin gene expression is inhibited by beta-adrenergic stimulation via protein kinase A in 3T3-L1 adipocytes.

Fasshauer M, Klein J, Neumann S, Eszlinger M, Paschke R. Fasshauer M, et al. Among authors: klein j. FEBS Lett. 2001 Oct 26;507(2):142-6. doi: 10.1016/s0014-5793(01)02960-x. FEBS Lett. 2001. PMID: 11684087 Free article.

4

Tumor necrosis factor alpha is a negative regulator of resistin gene expression and secretion in 3T3-L1 adipocytes.

Fasshauer M, Klein J, Neumann S, Eszlinger M, Paschke R. Fasshauer M, et al. Among authors: klein j. Biochem Biophys Res Commun. 2001 Nov 9;288(4):1027-31. doi: 10.1006/bbrc.2001.5874. Biochem Biophys Res Commun. 2001. PMID: 11689013

5

Direct effects of ciliary neurotrophic factor on brown adipocytes: evidence for a role in peripheral regulation of energy homeostasis.

Ott V, Fasshauer M, Dalski A, Klein HH, Klein J. Ott V, et al. Among authors: klein hh, klein j. J Endocrinol. 2002 May;173(2):R1-8. doi: 10.1677/joe.0.173r001. J Endocrinol. 2002. PMID: 12010646

6

Leptin secretion and negative autocrine crosstalk with insulin in brown adipocytes.

Kraus D, Fasshauer M, Ott V, Meier B, Jost M, Klein HH, Klein J. Kraus D, et al. Among authors: klein hh, klein j. J Endocrinol. 2002 Oct;175(1):185-91. doi: 10.1677/joe.0.1750185. J Endocrinol. 2002. PMID: 12379502

7

Interleukin (IL)-6 mRNA expression is stimulated by insulin, isoproterenol, tumour necrosis factor alpha, growth hormone, and IL-6 in 3T3-L1 adipocytes.

Fasshauer M, Klein J, Lossner U, Paschke R. Fasshauer M, et al. Among authors: klein j. Horm Metab Res. 2003 Mar;35(3):147-52. doi: 10.1055/s-2003-39075. Horm Metab Res. 2003. PMID: 12734774

8

Suppression of aquaporin adipose gene expression by isoproterenol, TNFalpha, and dexamethasone.

Fasshauer M, Klein J, Lossner U, Klier M, Kralisch S, Paschke R. Fasshauer M, et al. Among authors: klein j. Horm Metab Res. 2003 Apr;35(4):222-7. doi: 10.1055/s-2003-39478. Horm Metab Res. 2003. PMID: 12778365

9

GH is a positive regulator of tumor necrosis factor alpha-induced adipose related protein in 3T3-L1 adipocytes.

Fasshauer M, Klein J, Krahlisch S, Lössner U, Klier M, Blüher M, Paschke R. Fasshauer M, et al. Among authors: klein j. J Endocrinol. 2003 Sep;178(3):523-31. doi: 10.1677/joe.0.1780523. J Endocrinol. 2003. PMID: 12967343

10

Insulin resistance-inducing cytokines differentially regulate SOCS mRNA expression via growth factor- and Jak/Stat-signaling pathways in 3T3-L1 adipocytes.

Fasshauer M, Kralisch S, Klier M, Lossner U, Bluher M, Klein J, Paschke R. Fasshauer M, et al. Among authors: klein j. J Endocrinol. 2004 Apr;181(1):129-38. doi: 10.1677/joe.0.1810129. J Endocrinol. 2004. PMID: 15072573

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