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Lipoteichoic acid (LTA) of Streptococcus pneumoniae and Staphylococcus aureus activates immune cells via Toll-like receptor (TLR)-2, lipopolysaccharide-binding protein (LBP), and CD14, whereas TLR-4 and MD-2 are not involved.
Schröder NW, Morath S, Alexander C, Hamann L, Hartung T, Zähringer U, Göbel UB, Weber JR, Schumann RR. Schröder NW, et al. Among authors: morath s. J Biol Chem. 2003 May 2;278(18):15587-94. doi: 10.1074/jbc.M212829200. Epub 2003 Feb 19. J Biol Chem. 2003. PMID: 12594207 Free article.
Cytokine induction by purified lipoteichoic acids from various bacterial species--role of LBP, sCD14, CD14 and failure to induce IL-12 and subsequent IFN-gamma release.
Hermann C, Spreitzer I, Schröder NW, Morath S, Lehner MD, Fischer W, Schütt C, Schumann RR, Hartung T. Hermann C, et al. Among authors: morath s. Eur J Immunol. 2002 Feb;32(2):541-51. doi: 10.1002/1521-4141(200202)32:2<541::AID-IMMU541>3.0.CO;2-P. Eur J Immunol. 2002. PMID: 11828371 Free article.
Lipoteichoic acid (LTA) from Staphylococcus aureus stimulates human neutrophil cytokine release by a CD14-dependent, Toll-like-receptor-independent mechanism: Autocrine role of tumor necrosis factor-[alpha] in mediating LTA-induced interleukin-8 generation.
Hattar K, Grandel U, Moeller A, Fink L, Iglhaut J, Hartung T, Morath S, Seeger W, Grimminger F, Sibelius U. Hattar K, et al. Among authors: morath s. Crit Care Med. 2006 Mar;34(3):835-41. doi: 10.1097/01.ccm.0000202204.01230.44. Crit Care Med. 2006. PMID: 16521278
48 results