Bramlett HM - Search Results

1

Synuclein aggregation: possible role in traumatic brain injury.

Bramlett HM, Dietrich WD. Bramlett HM, et al. Exp Neurol. 2003 Nov;184(1):27-30. doi: 10.1016/s0014-4886(03)00401-1. Exp Neurol. 2003. PMID: 14637075 No abstract available.

2

Posttraumatic brain hypothermia provides protection from sensorimotor and cognitive behavioral deficits.

Bramlett HM, Green EJ, Dietrich WD, Busto R, Globus MY, Ginsberg MD. Bramlett HM, et al. J Neurotrauma. 1995 Jun;12(3):289-98. doi: 10.1089/neu.1995.12.289. J Neurotrauma. 1995. PMID: 7473803

3

Chronic histopathological consequences of fluid-percussion brain injury in rats: effects of post-traumatic hypothermia.

Bramlett HM, Dietrich WD, Green EJ, Busto R. Bramlett HM, et al. Acta Neuropathol. 1997 Feb;93(2):190-9. doi: 10.1007/s004010050602. Acta Neuropathol. 1997. PMID: 9039468

4

Hippocampally dependent and independent chronic spatial navigational deficits following parasagittal fluid percussion brain injury in the rat.

Bramlett HM, Green EJ, Dietrich WD. Bramlett HM, et al. Brain Res. 1997 Jul 11;762(1-2):195-202. doi: 10.1016/s0006-8993(97)00387-9. Brain Res. 1997. PMID: 9262173 Free article.

5

Temporal and regional patterns of axonal damage following traumatic brain injury: a beta-amyloid precursor protein immunocytochemical study in rats.

Bramlett HM, Kraydieh S, Green EJ, Dietrich WD. Bramlett HM, et al. J Neuropathol Exp Neurol. 1997 Oct;56(10):1132-41. doi: 10.1097/00005072-199710000-00007. J Neuropathol Exp Neurol. 1997. PMID: 9329457

6

Exacerbation of cortical and hippocampal CA1 damage due to posttraumatic hypoxia following moderate fluid-percussion brain injury in rats.

Bramlett HM, Green EJ, Dietrich WD. Bramlett HM, et al. J Neurosurg. 1999 Oct;91(4):653-9. doi: 10.3171/jns.1999.91.4.0653. J Neurosurg. 1999. PMID: 10507388

7

Secondary hypoxia following moderate fluid percussion brain injury in rats exacerbates sensorimotor and cognitive deficits.

Bramlett HM, Dietrich WD, Green EJ. Bramlett HM, et al. J Neurotrauma. 1999 Nov;16(11):1035-47. doi: 10.1089/neu.1999.16.1035. J Neurotrauma. 1999. PMID: 10595820

8

Chronic metabolic sequelae of traumatic brain injury: prolonged suppression of somatosensory activation.

Passineau MJ, Zhao W, Busto R, Dietrich WD, Alonso O, Loor JY, Bramlett HM, Ginsberg MD. Passineau MJ, et al. Among authors: bramlett hm. Am J Physiol Heart Circ Physiol. 2000 Sep;279(3):H924-31. doi: 10.1152/ajpheart.2000.279.3.H924. Am J Physiol Heart Circ Physiol. 2000. PMID: 10993751 Free article.

9

Delayed hemorrhagic hypotension exacerbates the hemodynamic and histopathologic consequences of traumatic brain injury in rats.

Matsushita Y, Bramlett HM, Kuluz JW, Alonso O, Dietrich WD. Matsushita Y, et al. Among authors: bramlett hm. J Cereb Blood Flow Metab. 2001 Jul;21(7):847-56. doi: 10.1097/00004647-200107000-00010. J Cereb Blood Flow Metab. 2001. PMID: 11435797

10

Neuropathological protection after traumatic brain injury in intact female rats versus males or ovariectomized females.

Bramlett HM, Dietrich WD. Bramlett HM, et al. J Neurotrauma. 2001 Sep;18(9):891-900. doi: 10.1089/089771501750451811. J Neurotrauma. 2001. PMID: 11565601

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